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p73α1, a p73 C-terminal isoform, regulates tumor suppression and the inflammatory response via Notch1

Proceedings of the National Academy of Sciences(2022)10.1073/pnas.2123202119Source: DataRank Database

p73α1, a p73 C-terminal isoform, regulates tumor suppression and the inflammatory response via Notch1 is a research paper published in Proceedings of the National Academy of Sciences (2022). On theSindex it has a DataRank of 0.345. It has been cited 9 times.

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0.345DataRank · unranked

Scored on demand from live citation data

Open Access9 citations

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Linked data & code

Exploring DNA Polymerase Eta as a Target to Overcome the Resistance to Platinum-based Drugs in NSCLC · University of California Office of the President

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›Methodology & internals

datarank_citation_only_1hop_v6· scope data_onlyMethodology

Pipeline:MetadataData-paper checkEnrichmentCitation networkScoring

Enrichment:Funding (13 grants)Impact (FWCI 0.65)OA: greenTopics (68)Linked Datasets (1)DataCite Reuse (1)IDs (PubMed, PMC, OpenAlex)SDGs (1)

FAIR Checklist

Context only (not used in score)

Findable (2/2)

Has DOI
Indexed in repositories

Accessible (1/2)

Open Access

Interoperable (2/2)

DataCite relations
Linked datasets

Reusable (0/3)

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DataRank Breakdown

Base Score 100%Citation Network 0%

Base Score Contribution

0.345

From this paper's citation signal

Citation Network Contribution

Citation network not refreshed for this result

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Authors (7)

Wensheng Yan ORCID,Xiangmudong Kong,Wenqiang Sun ORCID,Mingyi Chen ORCID,Jin Zhang ORCID

Live enrichment

PubMed:35617425 PMC:PMC9296871 OpenAlex:https://openalex.org/W4281569328

HHS | NIH | Office of Extramural Research, National Institutes of Health

Grant: CA081237

HHS | NIH | Office of Extramural Research, National Institutes of Health

Grant: CA224433

Tobacco-Related Disease Research Program

Grant: T31IP1727

HHS | NIH | National Cancer Institute

Grant: CA093373

UC | University of California, Davis

Grant: HL007013

NCI NIH HHS

Grant: P30 CA093373

NCI NIH HHS

Grant: R01 CA081237

NHLBI NIH HHS

Grant: T32 HL007013

NCI NIH HHS

Grant: R01 CA224433

National Institutes of Health

Grant: 5R01CA081237-18

Mechanism of P73-Dependent Tumor Suppression

National Institutes of Health

Grant: 1R01CA224433-01

The feedback loop between ferredoxin reductase and the p53 family in tumor suppression

National Institutes of Health

Grant: 3P30CA093373-07S2

Cancer Center Support Grant - P30

National Institutes of Health

Grant: 5T32HL007013-23

TRAINING IN COMPARATIVE LUNG BIOLOGY AND MEDICINE

FWCI

0.65

Citation Percentile

0.7%

Citation Trend

2022

2023

2024

2025

2026

greenLicense: cc-by-nc-nd

repositoryhttps://www.ncbi.nlm.nih.gov/pmc/articles/9296871

publisherhttps://pnas.org/doi/pdf/10.1073/pnas.2123202119

journalhttps://doi.org/10.1073/pnas.2123202119

repositoryhttps://pubmed.ncbi.nlm.nih.gov/35617425

Fields of Study

Cancer-related Molecular PathwaysRNA Interference and Gene DeliveryCancer Research and Treatments0301 basic medicine03 medical and health sciences0303 health sciences

MeSH Terms

Tumor Protein p73AnimalsDNA DamageExonsHumansInflammationNeoplasmsGenes, Tumor SuppressorMice, KnockoutProtein IsoformsCell Line, TumorMiceReceptor, Notch1Gene Knockout Techniques

Keywords

Gene knockdownGene isoformCell growthEctopic expressionBiologyCell biologyAlternative splicingCancer researchRegulatorExonEmbryonic stem cellKnockout mouseCell cultureMolecular biologyReceptorGeneGeneticsTumor suppressorp73P53 FamilyNotch1 PathwayP73 C-terminal Isoforms570Knockout610Cell LineGene Knockout TechniquesMiceCell Line, TumorNeoplasms2.1 Biological and endogenous factorsAnimalsHumansProtein IsoformsGenes, Tumor SuppressorReceptor, Notch1CancerInflammationMice, KnockoutNotch1TumorBiomedical and Clinical SciencesTumor Protein p73ExonsBiological SciencesGenesBiochemistry and Cell BiologyDNA Damage

Sustainable Development Goals

SDG 0: Good health and well-being

Exploring DNA Polymerase Eta as a Target to Overcome the Resistance to Platinum-based Drugs in NSCLC

10.17920/g96v7bUniversity of California Office of the President