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Anaplasma phagocytophilum effector EgeA facilitates infection by hijacking TANGO1 and SCFD1 from ER–Golgi exit sites to pathogen-occupied inclusions

Proceedings of the National Academy of Sciences(2024)10.1073/pnas.2405209121Source: DataRank Database

Anaplasma phagocytophilum effector EgeA facilitates infection by hijacking TANGO1 and SCFD1 from ER–Golgi exit sites to pathogen-occupied inclusions is a research paper published in Proceedings of the National Academy of Sciences (2024). On theSindex it has a DataRank of 0. It has been cited 7 times.

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Open Access7 citations

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datarank_citation_only_1hop_v6· scope data_onlyMethodology

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Enrichment:Funding (2 grants)Impact (FWCI 1.09)OA: hybridTopics (44)IDs (PubMed, PMC, OpenAlex)

FAIR Checklist

Context only (not used in score)

Findable (1/2)

Has DOI

Accessible (1/2)

Open Access

Interoperable (0/2)

Reusable (0/3)

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Authors (4)

Mingqun Lin ORCID,Libo Hou ORCID,Yasuko Rikihisa ORCID,Lidan Wang ORCID

Live enrichment

PubMed:39106308 PMC:PMC11331065 OpenAlex:https://openalex.org/W4401358587

HHS | NIH | NIAID | Division of Microbiology and Infectious Diseases

Grant: R01AI151065

National Institutes of Health

Grant: 5R01AI151065-04

Keys to prevent iron hijacking and infection by intracellular bacteria

FWCI

1.09

Citation Percentile

0.8%

Citation Trend

2025

2026

hybridLicense: cc-by-nc-nd

journalhttps://www.pnas.org/doi/pdf/10.1073/pnas.2405209121

publisherhttps://www.pnas.org/doi/pdf/10.1073/pnas.2405209121

publisherhttps://pnas.org/doi/pdf/10.1073/pnas.2405209121

journalhttps://doi.org/10.1073/pnas.2405209121

repositoryhttps://pubmed.ncbi.nlm.nih.gov/39106308

Fields of Study

Toxin Mechanisms and ImmunotoxinsTransgenic Plants and ApplicationsMosquito-borne diseases and control0301 basic medicine0303 health sciences03 medical and health sciencesAnaplasma phagocytophilumEndoplasmic ReticulumHumansGolgi ApparatusBacterial ProteinsInclusion BodiesAnimalsType IV Secretion SystemsHost-Pathogen Interactions

MeSH Terms

Type IV Secretion SystemsAnimalsBacterial ProteinsInclusion BodiesEndoplasmic ReticulumGolgi ApparatusHumansAnaplasma phagocytophilumHost-Pathogen Interactions

Keywords

Anaplasma phagocytophilumEffectorPathogenBiologyGolgi apparatusMicrobiologyCell biologyImmunologyHuman Granulocytic AnaplasmosisTango1Scfd1T4ss EffectorInclusion BodiesType IV Secretion SystemsBacterial ProteinsHost-Pathogen InteractionsHumansAnimalsBiological SciencesEndoplasmic Reticulum