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FAK in the nucleus prevents VSMC proliferation by promoting p27 and p21 expression via Skp2 degradation

Cardiovascular Research(2021)10.1093/cvr/cvab132Source: DataRank Database

FAK in the nucleus prevents VSMC proliferation by promoting p27 and p21 expression via Skp2 degradation is a research paper published in Cardiovascular Research (2021). On theSindex it has a DataRank of 0. It has been cited 36 times.

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Open Access36 citations

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›Methodology & internals

datarank_citation_only_1hop_v6· scope data_onlyMethodology

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Enrichment:Funding (7 grants)Impact (FWCI 3.41)OA: greenTopics (37)Linked Datasets (6)DataCite Reuse (6)IDs (PubMed, PMC, OpenAlex)

FAIR Checklist

Context only (not used in score)

Findable (2/2)

Has DOI
Indexed in repositories

Accessible (1/2)

Open Access

Interoperable (2/2)

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Reusable (0/3)

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Authors (4)

James M. Murphy ORCID,E. Ahn ORCID,Ssang‐Taek Lim ORCID,Kyuho Jeong ORCID

Live enrichment

PubMed:33839758 PMC:PMC8930076 OpenAlex:https://openalex.org/W3153065070

American Heart Association

Grant: 12SDG10970000

American Heart Association

Grant: 16GRNT30960007

National Institutes of Health

Grant: R01 CA190688

National Institutes of Health

Grant: R01 HL136432

Nikon A1 Confocal microscope was supported by the National Institutes of Health

Grant: S10RR027535

University of South Alabama

2019 College of Medicine Intramural grant from University of South Alabama

FWCI

3.41

Citation Percentile

0.9%

Citation Trend

2021

2022

2023

2024

2025

2026

greenLicense: https://academic.oup.com/journals/pages/open_access/funder_policies/chorus/standard_publication_model

repositoryhttps://www.ncbi.nlm.nih.gov/pmc/articles/8930076

publisherhttp://academic.oup.com/cardiovascres/advance-article-pdf/doi/10.1093/cvr/cvab132/37940511/cvab132.pdf

publisherhttps://academic.oup.com/cardiovascres/article-pdf/118/4/1150/42950171/cvab132.pdf

journalhttps://doi.org/10.1093/cvr/cvab132

Fields of Study

Cell Adhesion Molecules ResearchCellular Mechanics and InteractionsWnt/β-catenin signaling in development and cancer

MeSH Terms

Cells, CulturedMuscle, Smooth, VascularCyclin D1S-Phase Kinase-Associated ProteinsCell ProliferationCyclin-Dependent Kinase Inhibitor p21Cyclin-Dependent Kinase Inhibitor p27Focal Adhesion Protein-Tyrosine Kinases

Keywords

Cyclin-dependent kinaseFocal adhesionCell biologySKP2Cyclin D1CyclinCancer researchCyclin DVascular smooth muscleUbiquitin ligaseBiologyKinaseCell cycleSignal transductionUbiquitinApoptosisEndocrinologyBiochemistryp21CDKp27FAKVSMCVascular InjuryNeointimal HyperplasiaFzr1

Additional file 1 of G6PD maintains the VSMC synthetic phenotype and accelerates vascular neointimal hyperplasia by inhibiting the VDAC1–Bax-mediated mitochondrial apoptosis pathway

10.6084/m9.figshare.25566286.v1figshare

Additional file 1 of G6PD maintains the VSMC synthetic phenotype and accelerates vascular neointimal hyperplasia by inhibiting the VDAC1–Bax-mediated mitochondrial apoptosis pathway

10.6084/m9.figshare.25566286figshare

Additional file 2 of G6PD maintains the VSMC synthetic phenotype and accelerates vascular neointimal hyperplasia by inhibiting the VDAC1–Bax-mediated mitochondrial apoptosis pathway

10.6084/m9.figshare.25566391.v1figshare

Additional file 2 of G6PD maintains the VSMC synthetic phenotype and accelerates vascular neointimal hyperplasia by inhibiting the VDAC1–Bax-mediated mitochondrial apoptosis pathway

10.6084/m9.figshare.25566391figshare

Additional file 3 of G6PD maintains the VSMC synthetic phenotype and accelerates vascular neointimal hyperplasia by inhibiting the VDAC1–Bax-mediated mitochondrial apoptosis pathway

10.6084/m9.figshare.26703733.v1figshare

Additional file 3 of G6PD maintains the VSMC synthetic phenotype and accelerates vascular neointimal hyperplasia by inhibiting the VDAC1–Bax-mediated mitochondrial apoptosis pathway

10.6084/m9.figshare.26703733figshare