Down-regulation of AMPK/PPARδ signalling promotes endoplasmic reticulum stress-induced endothelial dysfunction in adult rat offspring exposed to maternal diabetes is a research paper published in Cardiovascular Research (2021). On theSindex it has a DataRank of 0. It has been cited 44 times.
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National Natural Science Foundation of China
Grant: 81700375
National Natural Science Foundation of China
Grant: 31730043
Basic and Frontier Research Program of Chongqing, China
Grant: cstc2017jcyjAX0153
Chongqing Science and health joint medical research
Grant: 2021MSXM020
National Key R&D Program of China
Grant: 2018YFC1312700
National Natural Science Foundation
Grant: 81721001
Program for Changjiang Scholars and Innovative Research Team in University
Grant: IRT1216
Program for Changjiang Scholars and Innovative Research Team in University
Grant: DK039308
Program for Changjiang Scholars and Innovative Research Team in University
Grant: HL074940
Program for Changjiang Scholars and Innovative Research Team in University
Grant: DK119652
NIDDK NIH HHS
Grant: R01 DK119652
NHLBI NIH HHS
Grant: P01 HL074940
NIDDK NIH HHS
Grant: R01 DK039308
National Institutes of Health
FWCI
10.33
Citation Percentile
1.0%
Citation Trend
Fields of Study
MeSH Terms
Keywords
Sustainable Development Goals
Additional file 1 of MiR-206 may regulate mitochondrial ROS contribute to the progression of Myocardial infarction via TREM1
Additional file 2 of MiR-206 may regulate mitochondrial ROS contribute to the progression of Myocardial infarction via TREM1
Additional file 1 of MiR-206 may regulate mitochondrial ROS contribute to the progression of Myocardial infarction via TREM1
Additional file 2 of MiR-206 may regulate mitochondrial ROS contribute to the progression of Myocardial infarction via TREM1
Additional file 3 of MiR-206 may regulate mitochondrial ROS contribute to the progression of Myocardial infarction via TREM1
Additional file 3 of MiR-206 may regulate mitochondrial ROS contribute to the progression of Myocardial infarction via TREM1