Emerging epigenetic therapies of cardiac fibrosis and remodelling in heart failure: from basic mechanisms to early clinical development is a research paper published in Cardiovascular Research (2022). On theSindex it has a DataRank of 0.650. It has been cited 75 times.
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Base Score Contribution
0.650
From this paper's citation signal
Citation Network Contribution
0
Citation network not refreshed for this result
This paper's DataRank is currently driven only by its base citation score. Citation network data was not refreshed for this result.
Learn more about DataRank methodology →National Institutes of Health
Grant: HL116848
National Institutes of Health
Grant: HL147558
National Institutes of Health
Grant: DK119594
National Institutes of Health
Grant: HL127240
National Institutes of Health
Grant: HL150225
National Institutes of Health
Grant: HL147463
American Heart Association
Grant: 19CDA34670044
ERC Advanced Grant REVERSE
Grant: DFG TRR267
ERC Advanced Grant REVERSE
Grant: CRC1470
NHLBI NIH HHS
Grant: F32 HL147463
National Institutes of Health
Grant: 1R01HL127240-01
Regulation of Chromatin Signaling in Heart Failure by BET Bromodomain Proteins
National Institutes of Health
Grant: 5F32HL147463-02
Exploring the Therapeutic Potential of BRD4 Extra-terminal Domain Inhibition in Cardiac Dysfunction and Remodeling. Fellow: Joshua Travers
National Institutes of Health
Grant: 5R01DK119594-03
Screening and Development of Small Molecule HDAC11 Inhibitors to Treat Obesity and Diabetes.
National Institutes of Health
Grant: 5R01HL147558-02
Deacetylase-Dependent Control of Diastolic Dysfunction and HFpEF
National Institutes of Health
Grant: 5R01HL150225-03
Small molecule therapies targeting chromatin architecture in heart failure
National Institutes of Health
Grant: 2R01HL116848-05A1
Regulation of Cardiac Signaling by Class I Histone Deacetylases
Singapore National Medical Research Council
Biomedical Research Council
FWCI
4.71
Citation Percentile
1.0%
Citation Trend
Fields of Study
MeSH Terms
Keywords
Sustainable Development Goals