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Expression of Concern: Hippocampal phosphorylated tau induced cognitive decline, dendritic spine loss and mitochondrial abnormalities in a mouse model of Alzheimer's disease

Human Molecular Genetics(2025)10.1093/hmg/ddaf098Source: DataRank Database

Expression of Concern: Hippocampal phosphorylated tau induced cognitive decline, dendritic spine loss and mitochondrial abnormalities in a mouse model of Alzheimer's disease is a research paper published in Human Molecular Genetics (2025). On theSindex it has a DataRank of 0.

N/A

0DataRank · unranked

Scored on demand from live citation data

Open Access0 citations

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›Methodology & internals

datarank_citation_only_1hop_v6· scope data_onlyMethodology

Pipeline:MetadataData-paper checkEnrichmentCitation networkScoring

Enrichment:Funding (3 grants)Impact (FWCI 0.00)OA: bronzeTopics (16)IDs (PubMed, PMC, OpenAlex)

FAIR Checklist

Context only (not used in score)

Findable (1/2)

Has DOI

Accessible (1/2)

Open Access

Interoperable (0/2)

Reusable (0/3)

FAIR checklist signals are shown for context only and do not affect DataRank scoring.

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Live enrichment

PubMed:40499000 PMC:PMC13031166 OpenAlex:https://openalex.org/W4411202120

NIH

Grant: AG042178

NIH

Grant: AG047812

NIH

Grant: NS105473

FWCI

0.00

Citation Percentile

0.1%

bronzeLicense: https://academic.oup.com/pages/standard-publication-reuse-rights

journalhttps://academic.oup.com/hmg/advance-article-pdf/doi/10.1093/hmg/ddaf098/63472762/ddaf098.pdf

publisherhttps://academic.oup.com/hmg/advance-article-pdf/doi/10.1093/hmg/ddaf098/63472762/ddaf098.pdf

publisherhttps://academic.oup.com/hmg/article-pdf/34/14/1253/63472762/ddaf098.pdf

journalhttps://doi.org/10.1093/hmg/ddaf098

repositoryhttps://pubmed.ncbi.nlm.nih.gov/40499000

Fields of Study

Neuroscience and Neuropharmacology ResearchMitochondrial Function and PathologyAlzheimer's disease research and treatments

Keywords

BiologyHippocampal formationDendritic spineCognitive declinePhosphorylationAlzheimer's diseaseNeuroscienceHippocampusCell biologyMitochondrionDiseaseDementiaInternal medicine