Pilot corpus: NIH-funded biomedical datasets. Any DOI can be scored on demand — network effects sharpen as coverage grows.

Locus-specific transcription silencing at the FHIT gene suppresses replication stress-induced copy number variant formation and associated replication delay

Nucleic Acids Research(2021)10.1093/nar/gkab559Source: DataRank Database

Locus-specific transcription silencing at the FHIT gene suppresses replication stress-induced copy number variant formation and associated replication delay is a research paper published in Nucleic Acids Research (2021). On theSindex it has a DataRank of 0. It has been cited 25 times.

N/A

0DataRank · unranked

Scored on demand from live citation data

Open Access25 citations

Download PDF

Cite:

Linked data & code

geo igsr uniprot

DataRank reads this dataset's downstream impact straight off the citation graph — no black box, no proprietary weighting. How is this computed?

›Methodology & internals

datarank_citation_only_1hop_v6· scope data_onlyMethodology

Pipeline:MetadataData-paper checkEnrichmentCitation networkScoring

Enrichment:Funding (9 grants)OA: goldTopics (58)IDs (PubMed, PMC)Annotations (3)

FAIR Checklist

Context only (not used in score)

Findable (1/2)

Has DOI

Accessible (1/2)

Open Access

Interoperable (0/2)

Reusable (0/3)

FAIR checklist signals are shown for context only and do not affect DataRank scoring.

Run a calibrated FAIR evaluation for this paper →

We only score data papers we can read in full — never from an abstract alone.

Authors (7)

Pamela E. Bennett‐Baker,Samreen Ahmed,Martin F. Arlt ORCID,Mats Ljungman ORCID,Thomas W. Glover ORCID

Live enrichment

PubMed:34181717 PMC:PMC8287918

National Institutes of Health

Grant: CA200731

National Institutes of Health

Grant: GM007544

National Institutes of Health

Grant: HG009382

NCI NIH HHS

Grant: R01 CA200731

NIGMS NIH HHS

Grant: T32 GM007544

NCI NIH HHS

Grant: P30 CA046592

NHGRI NIH HHS

Grant: UM1 HG009382

National Institutes of Health

Grant: 5R01CA200731-02

Extreme genomic instability at large transcribed genes: mechanisms and consequences for the cancer genome

National Institutes of Health

Grant: 5T32GM007544-32

Michigan Predoctoral Training Program in Genetics

goldLicense: CC BY NC

publisherhttp://academic.oup.com/nar/article-pdf/49/13/7507/39117049/gkab559.pdf

Europe_PMChttps://europepmc.org/articles/PMC8287918

Europe_PMChttps://europepmc.org/articles/PMC8287918?pdf=render

https://doi.org/10.1093/nar/gkab559

https://academic.oup.com/nar/article-pdf/49/13/7507/39117049/gkab559.pdf

Fields of Study

0301 basic medicine0303 health sciences03 medical and health sciencesAcid Anhydride HydrolasesAnimalsAphidicolinCell LineChromosome Fragile SitesDNA Copy Number VariationsDNA ReplicationGenetic LociHumansMiceMutationNeoplasm ProteinsPromoter Regions, GeneticR-Loop StructuresRibonuclease HStress, PhysiologicalTranscription, GeneticBis(5'-adenosyl)triphosphatase

MeSH Terms

Cell LineAnimalsHumansMiceAphidicolinAcid Anhydride HydrolasesNeoplasm ProteinsDNA ReplicationTranscription, GeneticMutationChromosome Fragile SitesRibonuclease HPromoter Regions, GeneticStress, PhysiologicalGenetic LociDNA Copy Number VariationsR-Loop StructuresBis(5'-adenosyl)triphosphatase

Keywords

DNA ReplicationBis(5'-adenosyl)triphosphataseDNA Copy Number VariationsTranscription, GeneticChromosome Fragile SitesRibonuclease HGenome Integrity, Repair and ReplicationAcid Anhydride HydrolasesCell LineNeoplasm ProteinsMiceAphidicolinGenetic LociStress, PhysiologicalMutationAnimalsHumansR-Loop StructuresPromoter Regions, Genetic