Pilot corpus: NIH-funded biomedical datasets. Any DOI can be scored on demand — network effects sharpen as coverage grows.

MicroDNA levels are dependent on MMEJ, repressed by c-NHEJ pathway, and stimulated by DNA damage

Nucleic Acids Research(2021)10.1093/nar/gkab984Source: DataRank Database

MicroDNA levels are dependent on MMEJ, repressed by c-NHEJ pathway, and stimulated by DNA damage is a research paper published in Nucleic Acids Research (2021). On theSindex it has a DataRank of 0. It has been cited 101 times.

N/A

0DataRank · unranked

Scored on demand from live citation data

Open Access101 citations

Download PDF

Cite:

Linked data & code

geo doi

DataRank reads this dataset's downstream impact straight off the citation graph — no black box, no proprietary weighting. How is this computed?

›Methodology & internals

datarank_citation_only_1hop_v6· scope data_onlyMethodology

Pipeline:MetadataData-paper checkEnrichmentCitation networkScoring

Enrichment:Funding (8 grants)Impact (FWCI 5.04)OA: goldTopics (40)Linked Datasets (16)DataCite Reuse (16)IDs (PubMed, PMC, OpenAlex)Annotations (2)

FAIR Checklist

Context only (not used in score)

Findable (2/2)

Has DOI
Indexed in repositories

Accessible (1/2)

Open Access

Interoperable (2/2)

DataCite relations
Linked datasets

Reusable (0/3)

FAIR checklist signals are shown for context only and do not affect DataRank scoring.

Run a calibrated FAIR evaluation for this paper →

We only score data papers we can read in full — never from an abstract alone.

Authors (7)

Pumoli Malapati,Yoshiyuki Shibata,Briana Wilson ORCID,Rebeka Eki ORCID,Mouadh Benamar

Live enrichment

PubMed:34718766 PMC:PMC8599734 OpenAlex:https://openalex.org/W3210471498

NIH

Grant: R01 CA060499

NIGMS

Grant: R01 GM135376

CTG-T32

Grant: CA009109

NCI NIH HHS

Grant: F30 CA254134

NCI NIH HHS

Grant: T32 CA009109

National Institutes of Health

Grant: 5R01CA060499-08

DNA REPLICATION

National Institutes of Health

Grant: 5T32CA009109-23

CANCER RESEARCH TRAINING IN MOLECULAR BIOLOGY

National Institutes of Health

Grant: 5R01GM135376-05

Regulation of re-replication in mammalian cells

FWCI

5.04

Citation Percentile

1.0%

Citation Trend

2022

2023

2024

2025

2026

goldLicense: cc-by

journalhttps://doi.org/10.1093/nar/gkab984

publisherhttps://doi.org/10.1093/nar/gkab984

publisherhttps://academic.oup.com/nar/article-pdf/49/20/11787/41161718/gkab984.pdf

repositoryhttps://pubmed.ncbi.nlm.nih.gov/34718766

repositoryhttps://www.ncbi.nlm.nih.gov/pmc/articles/8599734

Fields of Study

DNA Repair MechanismsCancer Genomics and DiagnosticsGenomics and Chromatin Dynamics0301 basic medicine0303 health sciences03 medical and health sciencesDNA DamageDNA End-Joining RepairDNA ReplicationDNA, CircularHEK293 CellsHeLa CellsHumans

MeSH Terms

DNA DamageDNA ReplicationDNA, CircularHeLa CellsHumansHEK293 CellsDNA End-Joining RepairHela Cells

Keywords

BiologyDNA repairDNADNA damageReplication protein AExtrachromosomal DNAMolecular biologyDNA replicationHomologous recombinationGeneticsGeneCell biologyPlasmidDNA-binding proteinDNA End-Joining RepairHEK293 CellsHumansDNA, CircularHeLa Cells

Additional file 1 of A distinct circular DNA profile intersects with proteome changes in the genotoxic stress-related hSOD1G93A model of ALS

10.6084/m9.figshare.24136275.v1figshare

Additional file 1 of A distinct circular DNA profile intersects with proteome changes in the genotoxic stress-related hSOD1G93A model of ALS

10.6084/m9.figshare.24136275figshare

Additional file 2 of A distinct circular DNA profile intersects with proteome changes in the genotoxic stress-related hSOD1G93A model of ALS

10.6084/m9.figshare.24136278.v1figshare

Additional file 2 of A distinct circular DNA profile intersects with proteome changes in the genotoxic stress-related hSOD1G93A model of ALS

10.6084/m9.figshare.24136278figshare

Additional file 3 of A distinct circular DNA profile intersects with proteome changes in the genotoxic stress-related hSOD1G93A model of ALS

10.6084/m9.figshare.24136284.v1figshare

Additional file 3 of A distinct circular DNA profile intersects with proteome changes in the genotoxic stress-related hSOD1G93A model of ALS

10.6084/m9.figshare.24136284figshare

Additional file 4 of A distinct circular DNA profile intersects with proteome changes in the genotoxic stress-related hSOD1G93A model of ALS

10.6084/m9.figshare.24136287.v1figshare

Additional file 4 of A distinct circular DNA profile intersects with proteome changes in the genotoxic stress-related hSOD1G93A model of ALS

10.6084/m9.figshare.24136287figshare

Additional file 5 of A distinct circular DNA profile intersects with proteome changes in the genotoxic stress-related hSOD1G93A model of ALS

10.6084/m9.figshare.24136293.v1figshare

Additional file 5 of A distinct circular DNA profile intersects with proteome changes in the genotoxic stress-related hSOD1G93A model of ALS

10.6084/m9.figshare.24136293figshare

Additional file 6 of A distinct circular DNA profile intersects with proteome changes in the genotoxic stress-related hSOD1G93A model of ALS

10.6084/m9.figshare.24136296.v1figshare

Additional file 6 of A distinct circular DNA profile intersects with proteome changes in the genotoxic stress-related hSOD1G93A model of ALS

10.6084/m9.figshare.24136296figshare

Additional file 7 of A distinct circular DNA profile intersects with proteome changes in the genotoxic stress-related hSOD1G93A model of ALS

10.6084/m9.figshare.24136299.v1figshare

Additional file 7 of A distinct circular DNA profile intersects with proteome changes in the genotoxic stress-related hSOD1G93A model of ALS

10.6084/m9.figshare.24136299figshare

Additional file 1 of Increased serum extrachromosomal circular DNA SORBS1circle level is associated with insulin resistance in patients with newly diagnosed type 2 diabetes mellitus

10.6084/m9.figshare.26666704.v1figshare

Additional file 1 of Increased serum extrachromosomal circular DNA SORBS1circle level is associated with insulin resistance in patients with newly diagnosed type 2 diabetes mellitus

10.6084/m9.figshare.26666704figshare

MicroDNA levels are dependent on MMEJ, repressed by c-NHEJ pathway, and stimulated by DNA damage

FAIR Checklist

Authors (7)

Funding (8 grants)

Impact

Open Access

Topics

Linked Datasets (16)

Annotations (Europe PMC)