The protection of Gα z -null NOD mice from hyperglycemia is sexually dimorphic and only partially β-cell autonomous is a research paper published in bioRxiv (Cold Spring Harbor Laboratory) (2021). On theSindex it has a DataRank of 0.
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National Institutes of Health
Grant: 3K01DK080845-02S1
G(alpha)Z signaling in insulin secretion and glucose tolerance
National Institutes of Health
Grant: 5P30CA014520-18
CANCER CENTER SUPPORT
National Institutes of Health
Grant: 5F31DK109698-02
Role of inhibitory G protein signaling in regulating beta-cell function and survival in the type 1 diabetic state
National Institutes of Health
Grant: 3R01DK102598-01S1
Molecular Mechanisms of Dysfunctional Prostaglandin Signaling in the Beta-Cell
Fields of Study
Keywords
Sustainable Development Goals