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Drug resistance mechanisms create targetable proteostatic vulnerabilities in Her2+ breast cancers

bioRxiv (Cold Spring Harbor Laboratory)(2021)10.1101/2021.08.18.456887Source: DataRank Database

Drug resistance mechanisms create targetable proteostatic vulnerabilities in Her2+ breast cancers is a research paper published in bioRxiv (Cold Spring Harbor Laboratory) (2021). On theSindex it has a DataRank of 0.

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datarank_citation_only_1hop_v6· scope data_onlyMethodology

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Enrichment:Funding (2 grants)OA: greenTopics (29)IDs (PubMed, OpenAlex)SDGs (2)

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Authors (5)

Lindsey E. Romick‐Rosendale ORCID,Miki Watanabe‐Chailland ORCID,Lisa M. Privette Vinnedge ORCID,Kakajan Komurov ORCID,Navneet Singh ORCID

Live enrichment

PubMed:19162232 OpenAlex:https://openalex.org/W3195728790

National Institutes of Health

Grant: 5R37CA218072-04

Mechanisms coupling DEK to oncogenesis

National Institutes of Health

Grant: 5R01CA193549-02

Exploiting proteotoxic stress in therapy-refractory HER2+ breast cancers

greenLicense: cc-by

repositoryhttps://www.biorxiv.org/content/biorxiv/early/2021/08/18/2021.08.18.456887.full.pdf

publisherhttps://syndication.highwire.org/content/doi/10.1101/2021.08.18.456887

repositoryhttps://doi.org/10.1101/2021.08.18.456887

repositoryhttps://doaj.org/article/d81778bc252946928d4caa112347ddcf

Fields of Study

Biochemical and Molecular ResearchPeptidase Inhibition and AnalysisEndoplasmic Reticulum Stress and Disease0301 basic medicine03 medical and health sciences

Keywords

Cancer researchUnfolded protein responseEndoplasmic reticulumOncogeneCell biologySignal transductionVemurafenibDrug resistanceCell growthBiologyKinaseCancerCellCell cycleBiochemistryGeneticsGlucoseScienceQRMedicineHumansNeoplasm Recurrence, LocalResearch Article

Sustainable Development Goals

SDG 3: 3. Good healthSDG 0: Good health and well-being