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Signaling network model of cardiomyocyte morphological changes in familial cardiomyopathy

bioRxiv (Cold Spring Harbor Laboratory)(2021)10.1101/2021.08.28.458032Source: DataRank Database

Signaling network model of cardiomyocyte morphological changes in familial cardiomyopathy is a research paper published in bioRxiv (Cold Spring Harbor Laboratory) (2021). On theSindex it has a DataRank of 0.

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›Methodology & internals

datarank_citation_only_1hop_v6· scope data_onlyMethodology

Pipeline:MetadataData-paper checkEnrichmentCitation networkScoring

Enrichment:Funding (3 grants)OA: greenTopics (47)IDs (PubMed, OpenAlex)SDGs (2)

FAIR Checklist

Context only (not used in score)

Findable (1/2)

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Accessible (1/2)

Open Access

Interoperable (0/2)

Reusable (0/3)

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Authors (7)

Muhammad Riaz ORCID,Stuart G. Campbell ORCID,Jeffrey H. Omens ORCID,Andrew D. McCulloch ORCID,Yibing Qyang ORCID

Live enrichment

PubMed:19767382 OpenAlex:https://openalex.org/W3198832909

National Institutes of Health

Grant: 1R01HL164783-01

Modulation of heart function by Muscle LIM protein-mediated mechanotransduction

National Institutes of Health

Grant: 5R01HL137100-03

Systems Biology of Hypertrophic Heart Disease from Molecular Pathways to Organ System

National Institutes of Health

Grant: 5R01HL137755-03

Systems Pharmacology Model for Spatial Control of Cardiac Fibrosis

greenLicense: cc-by-nd

repositoryhttps://www.biorxiv.org/content/biorxiv/early/2022/08/26/2021.08.28.458032.full.pdf

publisherhttps://syndication.highwire.org/content/doi/10.1101/2021.08.28.458032

repositoryhttps://doi.org/10.1101/2021.08.28.458032

repositoryhttps://escholarship.org/uc/item/796137g6

Fields of Study

Cardiomyopathy and Myosin StudiesCardiovascular Function and Risk FactorsCardiac Fibrosis and Remodeling0303 health sciences03 medical and health sciences

Keywords

TitinProtein kinase BHypertrophic cardiomyopathyDownregulation and upregulationPI3K/AKT/mTOR pathwayContext (archaeology)PhenotypeBiologyCalcineurinNFATCell biologySignal transductionMyocyteInternal medicineGeneticsMedicineGeneTransplantationSarcomereBiochemistryHeart FailureDCMMyocytesPatient-specific iPSC-CMsCardiomyopathyGenotype to phenotypeCardiomyopathy, HypertrophicHCMCardiac growthSignalingMicePhosphatidylinositol 3-KinasesHypertrophicFamilial cardiomyopathyAnimalsConnectinMyocytes, CardiacCalciumCardiomyopathiesCardiacProto-Oncogene Proteins c-aktInherited cardiomyopathy

Sustainable Development Goals

SDG 3: 3. Good healthSDG 0: Good health and well-being