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Distinct Mechanisms of Innate and Adaptive Immune Regulation Underlie Poor Oncologic Outcomes Associated with KRAS-TP53 Co-Alteration in Pancreatic Cancer

bioRxiv (Cold Spring Harbor Laboratory)(2022)10.1101/2022.05.01.490244Source: DataRank Database

Distinct Mechanisms of Innate and Adaptive Immune Regulation Underlie Poor Oncologic Outcomes Associated with KRAS-TP53 Co-Alteration in Pancreatic Cancer is a research paper published in bioRxiv (Cold Spring Harbor Laboratory) (2022). On theSindex it has a DataRank of 0.

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Authors (21)

Anna Bianchi ORCID,Iago De Castro Silva ORCID,Nilesh Deshpande ORCID,Long Cao ORCID,Siddharth Mehra ORCID

Live enrichment

PubMed:19965766 OpenAlex:https://openalex.org/W4225290451

National Institutes of Health

Grant: 3P30CA240139-02S3

The Sylvester Cancer Center Support Grant

National Institutes of Health

Grant: 5R01CA161976-03

Stat3 Signaling in Pancreas Cancer

National Institutes of Health

Grant: 5UL1TR002736-05

Miami Clinical and Translational Science Institute

greenLicense: Springer TDM

repositoryhttps://www.biorxiv.org/content/biorxiv/early/2022/05/02/2022.05.01.490244.full.pdf

publisherhttps://syndication.highwire.org/content/doi/10.1101/2022.05.01.490244

repositoryhttps://doi.org/10.1101/2022.05.01.490244

https://doi.org/10.1038/s41388-022-02368-w

Fields of Study

Pancreatic and Hepatic Oncology ResearchPhagocytosis and Immune RegulationImmune cells in cancer0301 basic medicine03 medical and health sciences

Keywords

KRASCancer researchInnate immune systemBiologyCancerTranscriptomeTumor microenvironmentPancreatic cancerImmune systemMyeloidImmunologyColorectal cancerPancreatic NeoplasmsProto-Oncogene Proteins p21(ras)MiceMutationAnimalsHumansAdenocarcinomaTumor Suppressor Protein p53Carcinoma, Pancreatic Ductal

Sustainable Development Goals

SDG 3: 3. Good healthSDG 0: No poverty