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Methylglyoxal-derived hydroimidazolone, MG-H1, increases food intake by altering tyramine signaling via the GATA transcription factor ELT-3 in Caenorhabditis elegans

bioRxiv (Cold Spring Harbor Laboratory)(2022)10.1101/2022.08.18.504374Source: DataRank Database

Methylglyoxal-derived hydroimidazolone, MG-H1, increases food intake by altering tyramine signaling via the GATA transcription factor ELT-3 in Caenorhabditis elegans is a research paper published in bioRxiv (Cold Spring Harbor Laboratory) (2022). On theSindex it has a DataRank of 0. It has been cited 1 time.

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Enrichment:Funding (2 grants)OA: greenTopics (18)IDs (PubMed, OpenAlex)SDGs (3)

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Findable (1/2)

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Accessible (1/2)

Open Access

Interoperable (0/2)

Reusable (0/3)

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Authors (11)

Jyotiska Chaudhuri ORCID,Durai Sellegounder ORCID,Amit Kumar Sahu ORCID,Sanjib Guha ORCID,Manish Chamoli ORCID

Live enrichment

PubMed:19910364 OpenAlex:https://openalex.org/W4294349308

National Institutes of Health

Grant: 5R01AG061165-02

Advanced glycation endproducts (AGEs) as metabolic by-products that mediate neurodegeneration.

National Institutes of Health

Grant: 5R01AG068288-02

Methylglyoxal drives astrocyte senescence to mediate neurodegeneration in Alzheimer's disease

greenLicense: cc-by

repositoryhttps://www.biorxiv.org/content/biorxiv/early/2022/08/19/2022.08.18.504374.full.pdf

publisherhttps://syndication.highwire.org/content/doi/10.1101/2022.08.18.504374

repositoryhttps://doi.org/10.1101/2022.08.18.504374

Fields of Study

Genetics, Aging, and Longevity in Model OrganismsAdvanced Glycation End Products researchBirth, Development, and Health0301 basic medicine0303 health sciences03 medical and health sciences

Keywords

MethylglyoxalCaenorhabditis elegansBiologyMutantTranscription factorGene knockdownDownregulation and upregulationBiochemistryCell biologyMaillard reactionGeneEnzyme

Sustainable Development Goals

SDG 2: 2. Zero hungerSDG 3: 3. Good healthSDG 0: Responsible consumption and production