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Demo corpus. Scores are computed on a select set of biomedical paper/datasets and may be inaccurate for papers outside this corpus — DataRank relies on network effects that improve with scale. We aim to expand this into a fully open resource pending additional funding.

Single-cell multi-cohort dissection of the schizophrenia transcriptome

(2022)10.1101/2022.08.31.22279406Source: DataRank Database

Single-cell multi-cohort dissection of the schizophrenia transcriptome is a dataset (2022). On theSindex it has a DataRank of 0.899, placing it in the top 42.8% of the data-sharing corpus. It has been cited 29 times, with 21 citing works in its 1-hop citation network. Its calibrated FAIR score is 15/100.

Top 43%percentile
0.899DataRank
0.899Top 43%
Dataset Open Access29 citations · base score 3.4
Cite:
datarank_citation_only_1hop_v6· scope data_onlyMethodology

Abstract

Schizophrenia is a prevalent mental illness with a high societal burden, complex pathophysiology, and diverse genetic and environmental etiology. Its complexity, polygenicity, and heterogeneity have hindered mechanistic elucidation and the search for new therapeutics. We present a single-cell dissection of schizophrenia-associated transcriptomic changes in the human prefrontal cortex across two independent cohorts, one deeply profiling 48 subjects (361,996 cells), and the other broadly profiling 92 subjects (106,761 cells). We identified 25 cell types that we used to produce a high-resolution atlas of schizophrenia-altered genes and pathways. Excitatory neurons were the most affected cell group, with transcriptional changes converging on neurodevelopment and synapse-related molecular pathways. Differentially expressed gene sets implicate a coherently expressed module of trans-acting regulatory factors involved in neurodevelopment and genetically associated with schizophrenia risk. Transcriptional alterations significantly overlapped with known genetic risk factors, suggesting convergence of rare and common genomic variants on reproducible neuronal population specific alterations in schizophrenia. The severity of transcriptional pathology segregated two populations of schizophrenia subjects in a manner consistent with the expression of specific transcriptional patterns marked by genes involved in synaptic function and chromatin dynamics. Our results provide a high-resolution single cell atlas linking transcriptomic changes within specific cell populations to etiological genetic risk factors, contextualizing established knowledge within the cytoarchitecture of the human cortex and facilitating mechanistic understanding of schizophrenia pathophysiology and heterogeneity.

Data sources & pipeline
Pipeline:MetadataData-paper checkEnrichmentCitation networkScoring
Enrichment:Pending

FAIR Checklist

Context only (not used in score)
Findable (1/2)
  • Has DOI
Accessible (1/2)
  • Open Access
Interoperable (0/2)
    Reusable (1/3)
    • Dataset classification

    FAIR checklist signals are shown for context only and do not affect DataRank scoring.

    15FAIR score
    F Findable
    20
    A Accessible
    30
    I Interoperable
    0
    R Reusable
    8
    Top 99% by FAIRLLM-assessed✓ full text read

    Calibrated FAIR score — a parallel quality metric, independent of the DataRank citation score. See the full evaluation →

    DataRank Breakdown

    Base Score 57%Citation Network 43%

    Base Score Contribution

    0.510

    From this paper's citation signal

    Citation Network Contribution

    0.388

    From 18 citing papers with measurable signal

    Learn more about DataRank methodology →

    Top 5 citers driving the network score

    Ranked by citation count — the same ordering the engine uses when summing log1p(Cq) over citers.

    Why this DataRank?

    DataRank blends this paper's own citation count with the influence of the papers that cite it. Here, roughly 57% comes from its base citations and 43% from the citation network (18 citing papers contributed measurable signal).

    Base score B(p)
    log1p(citation_count) — grows sub-linearly, so a paper with 1,000 citations is not 10× a paper with 100.
    Network N(p)
    Σ over citers of log1p(Cq) ÷ max(outdegreeq, 1). Being cited by a highly-cited paper with few references counts most.
    Damping factor d = 0.85
    DataRank = (1−d)·B(p) + d·N(p) — the two cards above are each already multiplied by their share.
    Self-citations excluded
    Citers sharing any OpenAlex author ID with this paper are filtered out before the network sum.

    Citers are pulled from OpenAlex sorted by cited_by_count:descand capped per paper, so when the cap binds we keep the highest-signal references and the score is reproducible across reruns.

    Read the full methodology →

    Click a node to highlight its connections. Use scroll to zoom. Drag to pan.

    Node colors:CenterData PaperData + Open AccessNon-dataSelected & links| Node size = percentile rank

    Authors (16)

    Shahin MohammadiORCID,Prashant S. EmaniORCID,Jose Davila-Velderrain,Sivan SubburajuORCID,Daniel Reed Tso