Pilot corpus: NIH-funded biomedical datasets. Any DOI can be scored on demand — network effects sharpen as coverage grows.

Autophagy disruption reduces mTORC1 activation leading to retinal ganglion cell neurodegeneration associated with glaucoma

bioRxiv (Cold Spring Harbor Laboratory)(2023)10.1101/2023.01.04.522687Source: DataRank Database

Autophagy disruption reduces mTORC1 activation leading to retinal ganglion cell neurodegeneration associated with glaucoma is a research paper published in bioRxiv (Cold Spring Harbor Laboratory) (2023). On theSindex it has a DataRank of 0. It has been cited 4 times.

N/A

0DataRank · unranked

Scored on demand from live citation data

Open Access4 citations

Download PDF

Cite:

DataRank reads this dataset's downstream impact straight off the citation graph — no black box, no proprietary weighting. How is this computed?

›Methodology & internals

datarank_citation_only_1hop_v6· scope data_onlyMethodology

Pipeline:MetadataData-paper checkEnrichmentCitation networkScoring

Enrichment:Funding (7 grants)OA: greenTopics (22)IDs (PubMed, OpenAlex)SDGs (1)

FAIR Checklist

Context only (not used in score)

Findable (1/2)

Has DOI

Accessible (1/2)

Open Access

Interoperable (0/2)

Reusable (0/3)

FAIR checklist signals are shown for context only and do not affect DataRank scoring.

Run a calibrated FAIR evaluation for this paper →

We only score data papers we can read in full — never from an abstract alone.

Authors (10)

Cátia Gomes ORCID,Yukihiro Shiga ORCID,Nicolás Belforte ORCID,Kirstin B. VanderWall,Sailee S. Lavekar ORCID

Live enrichment

PubMed:36711831 OpenAlex:https://openalex.org/W4313681525

Canadian Institutes of Health Research

Grant: unidentified

unidentified

National Institutes of Health

Grant: 1R01EY030838-01

Retinal Ganglion Cell Dendrite and Synapse Regeneration in Glaucoma: the Role of Insulin Signaling.

National Institutes of Health

Grant: 5R01EY033022-02

Establishing a human cellular model of retinal ganglion cell compartmentalization in neurodegeneration and neuroinflammation

National Institutes of Health

Grant: 5UL1TR002529-02

Indiana Clinical and Translational Sciences Institute

NEI NIH HHS

Grant: R01 EY030838

NEI NIH HHS

Grant: R01 EY033022

NCATS NIH HHS

Grant: UL1 TR002529

greenLicense: cc-by

repositoryhttps://www.biorxiv.org/content/biorxiv/early/2023/01/07/2023.01.04.522687.full.pdf

publisherhttps://syndication.highwire.org/content/doi/10.1101/2023.01.04.522687

repositoryhttps://doi.org/10.1101/2023.01.04.522687

repositoryhttps://pubmed.ncbi.nlm.nih.gov/36711831

Fields of Study

Autophagy in Disease and TherapyRetinal Development and DisordersRetinal Diseases and Treatments0301 basic medicine03 medical and health sciences

Keywords

AutophagyNeurodegenerationmTORC1Cell biologyPI3K/AKT/mTOR pathwayBiologyMechanistic target of rapamycinRetinal ganglion cellNeuroscienceSignal transductionMedicineRetinaInternal medicineGlaucomaRetinal ganglion cellsAutophagy dysfunctionArticle

Sustainable Development Goals

SDG 0: Good health and well-being