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Nuclear receptor signaling via NHR-49/MDT-15 regulates stress resilience and proteostasis in response to reproductive and metabolic cues

bioRxiv (Cold Spring Harbor Laboratory)(2023)10.1101/2023.04.25.537803Source: DataRank Database

Nuclear receptor signaling via NHR-49/MDT-15 regulates stress resilience and proteostasis in response to reproductive and metabolic cues is a research paper published in bioRxiv (Cold Spring Harbor Laboratory) (2023). On theSindex it has a DataRank of 0.165. It has been cited 2 times.

N/A
0.165DataRank · unranked

Scored on demand from live citation data

Open Access2 citations
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Methodology & internals
datarank_citation_only_1hop_v6· scope data_onlyMethodology
Pipeline:MetadataData-paper checkEnrichmentCitation networkScoring
Enrichment:Funding (3 grants)OA: greenTopics (14)IDs (PubMed, OpenAlex)

FAIR Checklist

Context only (not used in score)
Findable (1/2)
  • Has DOI
Accessible (1/2)
  • Open Access
Interoperable (0/2)
    Reusable (0/3)

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      DataRank Breakdown

      Base Score 100%Citation Network 0%

      Base Score Contribution

      0.165

      From this paper's citation signal

      Citation Network Contribution

      0

      Citation network not refreshed for this result

      This paper's DataRank is currently driven only by its base citation score. Citation network data was not refreshed for this result.

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      Authors (8)

      Live enrichment

      National Institutes of Health

      Grant: 5P01AG054407-04

      Proteostasis in Aging and Neurodegenerative Disease

      National Institutes of Health

      Grant: 1RF1AG057296-01

      Regulation of Peripheral Proteostasis

      National Institutes of Health

      Grant: 5T32AG020506-03

      Mechanisms of Aging and Dementia Training Program

      Fields of Study

      Genetics, Aging, and Longevity in Model OrganismsAdipose Tissue and MetabolismMitochondrial Function and Pathology

      Keywords

      ProteostasisCell biologyBiologyNuclear receptorLipid metabolismSignal transductionTranscription factorEndocrinologyGeneticsGeneArticle