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Voluntary wheel running mitigates disease in an Orai1 gain-of-function mouse model of tubular aggregate myopathy

bioRxiv (Cold Spring Harbor Laboratory)(2023)10.1101/2023.09.29.559036Source: DataRank Database

Voluntary wheel running mitigates disease in an Orai1 gain-of-function mouse model of tubular aggregate myopathy is a research paper published in bioRxiv (Cold Spring Harbor Laboratory) (2023). On theSindex it has a DataRank of 0.208. It has been cited 3 times.

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0.208DataRank · unranked

Scored on demand from live citation data

Open Access3 citations

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›Methodology & internals

datarank_citation_only_1hop_v6· scope data_onlyMethodology

Pipeline:MetadataData-paper checkEnrichmentCitation networkScoring

Enrichment:Funding (3 grants)OA: greenTopics (33)IDs (PubMed, OpenAlex)SDGs (1)

FAIR Checklist

Context only (not used in score)

Findable (1/2)

Has DOI

Accessible (1/2)

Open Access

Interoperable (0/2)

Reusable (0/3)

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DataRank Breakdown

Base Score 100%Citation Network 0%

Base Score Contribution

0.208

From this paper's citation signal

Citation Network Contribution

Citation network not refreshed for this result

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Authors (13)

Nan Zhao ORCID,Haley M. Orciuoli,Alice Brasile ORCID,Laura Pietrangelo ORCID,Miao He ORCID

Live enrichment

PubMed:37808709 OpenAlex:https://openalex.org/W4387221302

Telethon

Grant: GGP19231

National Institutes of Health

Grant: 1R01AR086382-01

Pathophysiology of Tubular Aggregate Myopathy

National Institutes of Health

Grant: 5R21AR081068-02

Characterization of the Exercise-induced Orai1 Proteome in Skeletal Muscle

greenLicense: cc-by-nc-nd

repositoryhttps://www.biorxiv.org/content/biorxiv/early/2023/09/29/2023.09.29.559036.full.pdf

publisherhttps://syndication.highwire.org/content/doi/10.1101/2023.09.29.559036

repositoryhttps://doi.org/10.1101/2023.09.29.559036

repositoryhttps://pubmed.ncbi.nlm.nih.gov/37808709

Fields of Study

Ion Channels and ReceptorsExercise and Physiological ResponsesIon channel regulation and function0303 health sciences03 medical and health sciences

Keywords

MyopathyEndoplasmic reticulumEndocrinologyInternal medicineSkeletal muscleChemistryMuscle weaknessGenetically modified mouseMedicineCell biologyBiologyTransgeneBiochemistryGeneMaleORAI1 ProteinArticleMiceDisease Models, AnimalSarcoplasmic ReticulumPhysical Conditioning, AnimalGain of Function MutationAnimalsCalciumStromal Interaction Molecule 1Muscle, Skeletalaggregate; exercise; mitochondria; myopathy; physiology; proteomics; sarcoplasmic reticulum; skeletal muscleMyopathies, Structural, Congenital

Sustainable Development Goals

SDG 0: Good health and well-being