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Type I IFN signaling in the absence of IRGM1 promotes M. tuberculosis replication in immune cells by suppressing T cell responses

bioRxiv (Cold Spring Harbor Laboratory)(2023)10.1101/2023.10.03.560720Source: DataRank Database

Type I IFN signaling in the absence of IRGM1 promotes M. tuberculosis replication in immune cells by suppressing T cell responses is a research paper published in bioRxiv (Cold Spring Harbor Laboratory) (2023). On theSindex it has a DataRank of 0. It has been cited 4 times.

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Open Access4 citations

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datarank_citation_only_1hop_v6· scope data_onlyMethodology

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Enrichment:Funding (10 grants)OA: greenTopics (49)IDs (PubMed, OpenAlex)Annotations (1)SDGs (2)

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Findable (1/2)

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Accessible (1/2)

Open Access

Interoperable (0/2)

Reusable (0/3)

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Authors (11)

Michael E. McNehlan,Yassin Mreyoud ORCID,Rachel L. Kinsella ORCID,Asya Smirnov ORCID,Chanchal Sur Chowdhury ORCID

Live enrichment

PubMed:37873329 OpenAlex:https://openalex.org/W4387359206

National Institutes of Health

Grant: 5P30CA091842-05

Core--Pharmacology Facility

National Institutes of Health

Grant: 5R01AI132697-02

Autophagy proteins in the immune response to Mycobacterium tuberculosis infection

National Institutes of Health

Grant: 5T32AI007172-37

Infectious Disease/Basic Microbial Pathogenic Mechanisms

National Institutes of Health

Grant: 1UL1TR002345-01

Washington University Institute of Clinical and Translational Sciences

National Institutes of Health

Grant: 5U19AI142784-03

Autophagy Modulators as Novel Broad-Spectrum Anti-Infective Agents

NIAID NIH HHS

Grant: U19 AI142784

NCI NIH HHS

Grant: P30 CA091842

NIAID NIH HHS

Grant: T32 AI007172

NCATS NIH HHS

Grant: UL1 TR002345

NIAID NIH HHS

Grant: R01 AI132697

greenLicense: cc-by-nc

repositoryhttps://www.biorxiv.org/content/biorxiv/early/2023/10/05/2023.10.03.560720.full.pdf

publisherhttps://syndication.highwire.org/content/doi/10.1101/2023.10.03.560720

repositoryhttps://doi.org/10.1101/2023.10.03.560720

repositoryhttps://pubmed.ncbi.nlm.nih.gov/37873329

Fields of Study

Tuberculosis Research and Epidemiologyinterferon and immune responsesMycobacterium research and diagnosis0301 basic medicine0303 health sciences03 medical and health sciences

Keywords

AutophagyImmune systemMycobacterium tuberculosisBiologyCell biologyInterferonIn vivoT cellSignal transductionCellCell typeMacrophageTuberculosisMicrobiologyImmunologyIn vitroMedicineApoptosisBiochemistryGeneticsPathologyMice, KnockoutNeutrophilsT-LymphocytesLymphocyte ActivationArticleMice, Inbred C57BLMiceDisease Models, AnimalGTP-Binding ProteinsInterferon Type IMacrophages, AlveolarAnimalsHumansCells, CulturedIFN-γIRGM1IRGM3inflammationlunglymph nodeneutrophiltype I interferon

Sustainable Development Goals

SDG 3: 3. Good healthSDG 0: Good health and well-being

Type I IFN signaling in the absence of IRGM1 promotes M. tuberculosis replication in immune cells by suppressing T cell responses

FAIR Checklist

Authors (11)

Funding (10 grants)

Open Access

Topics

Annotations (Europe PMC)