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Comparison of a novel potentiator of CFTR channel activity to ivacaftor in ameliorating mucostasis caused by cigarette smoke in primary human bronchial airway epithelial cells

bioRxiv (Cold Spring Harbor Laboratory)(2024)10.1101/2024.03.01.582742Source: DataRank Database

Comparison of a novel potentiator of CFTR channel activity to ivacaftor in ameliorating mucostasis caused by cigarette smoke in primary human bronchial airway epithelial cells is a research paper published in bioRxiv (Cold Spring Harbor Laboratory) (2024). On theSindex it has a DataRank of 0.

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Enrichment:Funding (3 grants)OA: greenTopics (34)IDs (PubMed, OpenAlex)SDGs (2)

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Authors (15)

Jia Xin Jiang,Paul D. W. Eckford,Mohabir Ramjeesingh,Canhui Li ORCID,Ling Jun Huan

Live enrichment

PubMed:38496440 OpenAlex:https://openalex.org/W4392384430

National Institutes of Health

Grant: 3P30DK072482-03S3

UAB CF Research and Translation Core Center

National Institutes of Health

Grant: 5R35HL135816-04

Translational Program in CFTR-Related Airway Diseases

NIDDK NIH HHS

Grant: P30 DK072482

greenLicense: CC BY

repositoryhttps://www.biorxiv.org/content/biorxiv/early/2024/03/04/2024.03.01.582742.full.pdf

publisherhttps://syndication.highwire.org/content/doi/10.1101/2024.03.01.582742

repositoryhttps://doi.org/10.1101/2024.03.01.582742

repositoryhttps://pubmed.ncbi.nlm.nih.gov/38496440

Fields of Study

Cystic Fibrosis Research AdvancesNeonatal Respiratory Health ResearchIon channel regulation and function0301 basic medicine0303 health sciences03 medical and health sciences

Keywords

IvacaftorPotentiatorCystic fibrosisMucociliary clearanceMucusCOPDAirwayMedicineChemistryLungCystic fibrosis transmembrane conductance regulatorPharmacologyInternal medicineBiologyAnesthesiaRC705-779ResearchEpithelial CellsBronchiRespiratory MucosaQuinolonesAminophenolsDiseases of the respiratory systemHEK293 CellsSmokeHumansChloride Channel AgonistsCells, Cultured

Sustainable Development Goals

SDG 3: 3. Good healthSDG 0: Good health and well-being