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Attenuation of ATM signaling by ROS delays replicative senescence at physiological oxygen

bioRxiv (Cold Spring Harbor Laboratory)(2024)10.1101/2024.06.24.600514Source: DataRank Database

Attenuation of ATM signaling by ROS delays replicative senescence at physiological oxygen is a research paper published in bioRxiv (Cold Spring Harbor Laboratory) (2024). On theSindex it has a DataRank of 0. It has been cited 5 times.

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Open Access5 citations

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›Methodology & internals

datarank_citation_only_1hop_v6· scope data_onlyMethodology

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Enrichment:Funding (4 grants)OA: greenTopics (29)IDs (PubMed, OpenAlex)

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Open Access

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Authors (6)

Kaori K. Takai,Railia R. Gabbasova,Henry Sanford,Ekaterina V. Vinogradova ORCID,Titia de Lange ORCID

Live enrichment

PubMed:38979390 OpenAlex:https://openalex.org/W4400027169

National Institutes of Health

Grant: 2R01AG016642-21A1

Mechanism of the telomeric proliferation limit

National Institutes of Health

Grant: 1R35CA210036-01

Genome instability in cancer: telomeres and DNA repair

NIA NIH HHS

Grant: R01 AG016642

NCI NIH HHS

Grant: R35 CA210036

greenLicense: cc-by-nc-nd

repositoryhttps://doi.org/10.1101/2024.06.24.600514

publisherhttps://syndication.highwire.org/content/doi/10.1101/2024.06.24.600514

repositoryhttps://pubmed.ncbi.nlm.nih.gov/38979390

repositoryhttps://www.ncbi.nlm.nih.gov/pmc/articles/11230194

Fields of Study

Telomeres, Telomerase, and SenescenceMitochondrial Function and PathologyGenetics, Aging, and Longevity in Model Organisms0303 health sciences03 medical and health sciences

Keywords

SenescenceTelomereShelterinCell biologyTelomeraseBiologyCarcinogenesisSuppressorGeneticsGeneDNA-binding proteinTranscription factorAtaxia Telangiectasia Mutated ProteinsFibroblastsArticleOxygenHumansDNA Breaks, Double-StrandedReactive Oxygen SpeciesCellular SenescenceCells, CulturedTelomere ShorteningSignal TransductionCell Proliferation