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Modeling presynaptic inhibition by the amyloid precursor protein demonstrates one potential mechanism for preventing runaway synaptic modification in Alzheimer’s disease

bioRxiv (Cold Spring Harbor Laboratory)(2025)10.1101/2025.08.11.669740Source: DataRank Database

Modeling presynaptic inhibition by the amyloid precursor protein demonstrates one potential mechanism for preventing runaway synaptic modification in Alzheimer’s disease is a research paper published in bioRxiv (Cold Spring Harbor Laboratory) (2025). On theSindex it has a DataRank of 0.

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datarank_citation_only_1hop_v6· scope data_onlyMethodology

Pipeline:MetadataData-paper checkEnrichmentCitation networkScoring

Enrichment:Funding (9 grants)OA: greenTopics (27)IDs (PubMed, OpenAlex)

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Open Access

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Authors (3)

Michael E. Hasselmo ORCID,Heather C. Rice ORCID,Dylan Barber

Live enrichment

PubMed:21097893 OpenAlex:https://openalex.org/W4413340003

NIH

Grant: R01 MH120073

NIH

Grant: R01 MH60013

NIH

Grant: R01 MH052090

NIH

Grant: R35 GM142726

Grant: MURI N00014-16-1-2832

Grant: MURI N00014-19-1-2571

Grant: DURIP N00014-17-1-2304

National Institutes of Health

Grant: 7R35GM142726-04

Mechanisms of APP ectodomain function

National Institutes of Health

Grant: 5R01MH120073-05

Egocentric and Allocentric Spatial Coding in Cortex

greenLicense: cc-by-nc-nd

repositoryhttps://www.biorxiv.org/content/biorxiv/early/2025/08/15/2025.08.11.669740.full.pdf

publisherhttps://syndication.highwire.org/content/doi/10.1101/2025.08.11.669740

repositoryhttps://doi.org/10.1101/2025.08.11.669740

https://doi.org/10.1002/alz.70748

Fields of Study

Alzheimer's disease research and treatmentsNeuroscience and Neuropharmacology ResearchComputational Drug Discovery Methods03 medical and health sciences0302 clinical medicine

Keywords

Mechanism (biology)NeuroscienceAmyloid (mycology)DiseaseAmyloid precursor proteinAmyloid βAlzheimer's diseaseChemistryBiologyMedicineInternal medicinePhysicsAmyloid beta-Protein PrecursorReceptors, GABA-BAlzheimer DiseaseSynapsesModels, NeurologicalLong-Term PotentiationPresynaptic TerminalsHumansComputer SimulationResearch Article