Association of birth mode of delivery with infant faecal microbiota, potential pathobionts, and short chain fatty acids: a longitudinal study over the first year of life is a research paper published in BJOG An International Journal of Obstetrics & Gynaecology (2020). On theSindex it has a DataRank of 0. It has been cited 56 times.
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National Heart, Lung, and Blood Institute
Grant: K01HL141589
National Institutes of Health
Grant: R01DK094841
National Institutes of Health
Grant: T32 HL007024
Mid-Atlantic Nutrition Obesity Research Center
Grant: P30DK0372488
National Institutes of Health
Grant: 7K01HL141589-06
The Effects of Diet on the Gut Microbiome and Short Chain Fatty Acids
National Institutes of Health
Grant: 5T32HL007024-25
CARDIOVASCULAR EPIDEMIOLOGY INSTITUTIONAL TRAINING
National Institutes of Health
Grant: 5R01DK094841-03
Early child care and risk of obesity
FWCI
3.13
Citation Percentile
0.9%
Citation Trend
Fields of Study
MeSH Terms
Keywords
Additional file 1 of Clostridioides difficile infection in infants: a case report and literature review
Additional file 1 of Clostridioides difficile infection in infants: a case report and literature review
Additional file 9 of Gut barrier-microbiota imbalances in early life lead to higher sensitivity to inflammation in a murine model of C-section delivery
Additional file 9 of Gut barrier-microbiota imbalances in early life lead to higher sensitivity to inflammation in a murine model of C-section delivery
Additional file 10 of Gut barrier-microbiota imbalances in early life lead to higher sensitivity to inflammation in a murine model of C-section delivery
Additional file 10 of Gut barrier-microbiota imbalances in early life lead to higher sensitivity to inflammation in a murine model of C-section delivery
Additional file 5 of Gut barrier-microbiota imbalances in early life lead to higher sensitivity to inflammation in a murine model of C-section delivery
Additional file 8 of Gut barrier-microbiota imbalances in early life lead to higher sensitivity to inflammation in a murine model of C-section delivery
Additional file 7 of Gut barrier-microbiota imbalances in early life lead to higher sensitivity to inflammation in a murine model of C-section delivery
Additional file 6 of Gut barrier-microbiota imbalances in early life lead to higher sensitivity to inflammation in a murine model of C-section delivery
Additional file 7 of Gut barrier-microbiota imbalances in early life lead to higher sensitivity to inflammation in a murine model of C-section delivery
Additional file 8 of Gut barrier-microbiota imbalances in early life lead to higher sensitivity to inflammation in a murine model of C-section delivery
Additional file 3 of Gut barrier-microbiota imbalances in early life lead to higher sensitivity to inflammation in a murine model of C-section delivery
Additional file 6 of Gut barrier-microbiota imbalances in early life lead to higher sensitivity to inflammation in a murine model of C-section delivery
Additional file 4 of Gut barrier-microbiota imbalances in early life lead to higher sensitivity to inflammation in a murine model of C-section delivery
Additional file 4 of Gut barrier-microbiota imbalances in early life lead to higher sensitivity to inflammation in a murine model of C-section delivery
Additional file 5 of Gut barrier-microbiota imbalances in early life lead to higher sensitivity to inflammation in a murine model of C-section delivery
Additional file 1 of Gut barrier-microbiota imbalances in early life lead to higher sensitivity to inflammation in a murine model of C-section delivery
Additional file 1 of Gut barrier-microbiota imbalances in early life lead to higher sensitivity to inflammation in a murine model of C-section delivery
Additional file 2 of Gut barrier-microbiota imbalances in early life lead to higher sensitivity to inflammation in a murine model of C-section delivery