Mice with a specific deficiency of Pfkfb3 in myeloid cells are protected from hypoxia‐induced pulmonary hypertension is a research paper published in British Journal of Pharmacology (2020). On theSindex it has a DataRank of 0. It has been cited 51 times.
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National Natural Science Foundation of China
Grant: 81870324
National Natural Science Foundation of China
Grant: 82070461
American Heart Association
Grant: 19POST34430119
American Heart Association
Grant: 19TPA34910043
China Postdoctoral Science Foundation
Grant: 2020M670051
China Postdoctoral Science Foundation
Grant: 2020M680003
National Institutes of Health
Grant: R01HL134934
National Institutes of Health
Grant: R01EY030500
National Institutes of Health
Grant: R01HL138410
National Institutes of Health
Grant: R01HL142097
FWCI
3.62
Citation Percentile
0.9%
Citation Trend
Fields of Study
MeSH Terms
Keywords
Additional file 1 of Tubular epithelial cell-derived extracellular vesicles induce macrophage glycolysis by stabilizing HIF-1α in diabetic kidney disease
Additional file 1 of Tubular epithelial cell-derived extracellular vesicles induce macrophage glycolysis by stabilizing HIF-1α in diabetic kidney disease
Additional file 2 of Tubular epithelial cell-derived extracellular vesicles induce macrophage glycolysis by stabilizing HIF-1α in diabetic kidney disease
Additional file 2 of Tubular epithelial cell-derived extracellular vesicles induce macrophage glycolysis by stabilizing HIF-1α in diabetic kidney disease
Additional file 3 of Tubular epithelial cell-derived extracellular vesicles induce macrophage glycolysis by stabilizing HIF-1α in diabetic kidney disease
Additional file 3 of Tubular epithelial cell-derived extracellular vesicles induce macrophage glycolysis by stabilizing HIF-1α in diabetic kidney disease
Additional file 4 of Tubular epithelial cell-derived extracellular vesicles induce macrophage glycolysis by stabilizing HIF-1α in diabetic kidney disease
Additional file 4 of Tubular epithelial cell-derived extracellular vesicles induce macrophage glycolysis by stabilizing HIF-1α in diabetic kidney disease