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Conditional deletion of the multiple sclerosis susceptibility gene ATXN1 identifies cell‐autonomous effects in the B‐cell compartment

FEBS Journal(2025)10.1111/febs.70328Source: DataRank Database

Conditional deletion of the multiple sclerosis susceptibility gene ATXN1 identifies cell‐autonomous effects in the B‐cell compartment is a research paper published in FEBS Journal (2025). On theSindex it has a DataRank of 0. It has been cited 2 times.

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0DataRank · unranked

Scored on demand from live citation data

Open Access2 citations

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›Methodology & internals

datarank_citation_only_1hop_v6· scope data_onlyMethodology

Pipeline:MetadataData-paper checkEnrichmentCitation networkScoring

Enrichment:Funding (3 grants)Impact (FWCI 1.12)OA: hybridTopics (41)IDs (PubMed, PMC, OpenAlex)

FAIR Checklist

Context only (not used in score)

Findable (1/2)

Has DOI

Accessible (1/2)

Open Access

Interoperable (0/2)

Reusable (0/3)

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Authors (4)

Rachael Riley Denbrock,Cindy Carolina Martines,Alessandro Didonna ORCID,Jonathan J. Carver

Live enrichment

PubMed:41224709 PMC:PMC12958106 OpenAlex:https://openalex.org/W4416157697

National Institutes of Health

Grant: R03NS131908

East Carolina University

FWCI

1.12

Citation Percentile

0.8%

Citation Trend

2026

hybridLicense: cc-by-nc-nd

journalhttps://onlinelibrary.wiley.com/doi/pdfdirect/10.1111/febs.70328

publisherhttps://onlinelibrary.wiley.com/doi/pdfdirect/10.1111/febs.70328

publisherhttps://febs.onlinelibrary.wiley.com/doi/pdf/10.1111/febs.70328

publisherhttps://febs.onlinelibrary.wiley.com/doi/full-xml/10.1111/febs.70328

journalhttps://doi.org/10.1111/febs.70328

Fields of Study

Genetic Neurodegenerative DiseasesAutoimmune Neurological Disorders and TreatmentsMultiple Sclerosis Research StudiesAnimalsAtaxin-1B-LymphocytesMiceEncephalomyelitis, Autoimmune, ExperimentalMultiple SclerosisMice, KnockoutLymphocyte ActivationCell ProliferationT-LymphocytesDisease Models, AnimalMice, Inbred C57BLFemaleGene Deletion

MeSH Terms

Ataxin-1AnimalsB-LymphocytesDisease Models, AnimalEncephalomyelitis, Autoimmune, ExperimentalFemaleLymphocyte ActivationMice, Inbred C57BLMultiple SclerosisT-LymphocytesGene DeletionMice, KnockoutCell ProliferationMice

Keywords

Experimental autoimmune encephalomyelitisMultiple sclerosisCompartment (ship)Autoimmune diseasePhenotypeGeneAutoimmunityPathogenesisB cellsAtaxin‐1