Macrophage‐derived MMP‐9 enhances the progression of atherosclerotic lesions and vascular calcification in transgenic rabbits is a research paper published in Journal of Cellular and Molecular Medicine (2020). On theSindex it has a DataRank of 0. It has been cited 82 times.
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Japan Society for the Promotion of Science
Grant: 15H04718
Japan Society for the Promotion of Science
Grant: 2017JZ028
National Natural Science Foundation of China
Grant: 81570392
National Natural Science Foundation of China
Grant: 81770457
Foundation for the National Institutes of Health
Grant: R01HL117491
Foundation for the National Institutes of Health
Grant: R01HL129778
National Institutes of Health
Grant: 5R01HL129778-03
MPO, HDL Dysfunction and Cardiovascular Disease
National Institutes of Health
Grant: 5R01HL117491-03
CETP and HDL Function in Cardiovascular Diseases
Fields of Study
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Keywords
Additional file 1 of Identification of core genes associated with the anti-atherosclerotic effects of Salvianolic acid B and immune cell infiltration characteristics using bioinformatics analysis
Additional file 1 of Identification of core genes associated with the anti-atherosclerotic effects of Salvianolic acid B and immune cell infiltration characteristics using bioinformatics analysis
Additional file 2 of Identification of core genes associated with the anti-atherosclerotic effects of Salvianolic acid B and immune cell infiltration characteristics using bioinformatics analysis
Additional file 2 of Identification of core genes associated with the anti-atherosclerotic effects of Salvianolic acid B and immune cell infiltration characteristics using bioinformatics analysis
Additional file 3 of Identification of core genes associated with the anti-atherosclerotic effects of Salvianolic acid B and immune cell infiltration characteristics using bioinformatics analysis
Additional file 3 of Identification of core genes associated with the anti-atherosclerotic effects of Salvianolic acid B and immune cell infiltration characteristics using bioinformatics analysis
Additional file 4 of Identification of core genes associated with the anti-atherosclerotic effects of Salvianolic acid B and immune cell infiltration characteristics using bioinformatics analysis
Additional file 4 of Identification of core genes associated with the anti-atherosclerotic effects of Salvianolic acid B and immune cell infiltration characteristics using bioinformatics analysis
Additional file 5 of Identification of core genes associated with the anti-atherosclerotic effects of Salvianolic acid B and immune cell infiltration characteristics using bioinformatics analysis
Additional file 5 of Identification of core genes associated with the anti-atherosclerotic effects of Salvianolic acid B and immune cell infiltration characteristics using bioinformatics analysis
Additional file 6 of Identification of core genes associated with the anti-atherosclerotic effects of Salvianolic acid B and immune cell infiltration characteristics using bioinformatics analysis
Additional file 6 of Identification of core genes associated with the anti-atherosclerotic effects of Salvianolic acid B and immune cell infiltration characteristics using bioinformatics analysis
Additional file 1 of Nobiletin alleviates atherosclerosis by inhibiting lipid uptake via the PPARG/CD36 pathway
Additional file 1 of Nobiletin alleviates atherosclerosis by inhibiting lipid uptake via the PPARG/CD36 pathway
Additional file 4 of Nobiletin alleviates atherosclerosis by inhibiting lipid uptake via the PPARG/CD36 pathway
Additional file 4 of Nobiletin alleviates atherosclerosis by inhibiting lipid uptake via the PPARG/CD36 pathway
Additional file 3 of Nobiletin alleviates atherosclerosis by inhibiting lipid uptake via the PPARG/CD36 pathway
Additional file 5 of Nobiletin alleviates atherosclerosis by inhibiting lipid uptake via the PPARG/CD36 pathway
Additional file 5 of Nobiletin alleviates atherosclerosis by inhibiting lipid uptake via the PPARG/CD36 pathway
Additional file 2 of Nobiletin alleviates atherosclerosis by inhibiting lipid uptake via the PPARG/CD36 pathway