Aβ deposition is associated with increases in soluble and phosphorylated tau that precede a positive Tau PET in Alzheimer’s disease is a research paper published in Science Advances (2020). On theSindex it has a DataRank of 0. It has been cited 374 times.
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NIA NIH HHS
Grant: P30 AG062422
NINDS NIH HHS
Grant: U54 NS092089
NIA NIH HHS
Grant: U01 AG045390
NIA NIH HHS
Grant: U19 AG063911
NIA NIH HHS
Grant: P01 AG019724
NIA NIH HHS
Grant: R01 AG038791
NIA NIH HHS
Grant: K24 AG045333
Medical Research Council
Grant: UKDRI-1003
Parkinson Research Foundation
Torsten Söderberg Foundation
Swedish Medical Association
European Research Council
Marianne and Marcus Wallenberg Foundation
Strategic Research Area MultiPark
Swedish Alzheimer Foundation
the Knut and Alice Wallenberg Foundation
Skåne University Hospital Foundation
Konung Gustaf V:s och Drottning Victorias Frimurarestiftelse
Swedish Research Council Formas
Medical Faculty at Lund University
Parkinson foundation of Sweden
Region Skåne
Swedish Brain Foundation
FWCI
23.44
Citation Percentile
1.0%
Citation Trend
Fields of Study
MeSH Terms
Keywords
Sustainable Development Goals
Additional file 1 of Regional correlation of biochemical measures of amyloid and tau phosphorylation in the brain
Additional file 1 of Regional correlation of biochemical measures of amyloid and tau phosphorylation in the brain
Additional file 1 of Longitudinal amyloid and tau accumulation in autosomal dominant Alzheimer’s disease: findings from the Colombia-Boston (COLBOS) biomarker study
Additional file 1 of Longitudinal amyloid and tau accumulation in autosomal dominant Alzheimer’s disease: findings from the Colombia-Boston (COLBOS) biomarker study
Additional file 2 of Longitudinal amyloid and tau accumulation in autosomal dominant Alzheimer’s disease: findings from the Colombia-Boston (COLBOS) biomarker study
Additional file 2 of Longitudinal amyloid and tau accumulation in autosomal dominant Alzheimer’s disease: findings from the Colombia-Boston (COLBOS) biomarker study
Additional file 3 of Longitudinal amyloid and tau accumulation in autosomal dominant Alzheimer’s disease: findings from the Colombia-Boston (COLBOS) biomarker study
Additional file 3 of Longitudinal amyloid and tau accumulation in autosomal dominant Alzheimer’s disease: findings from the Colombia-Boston (COLBOS) biomarker study
Additional file 4 of Longitudinal amyloid and tau accumulation in autosomal dominant Alzheimer’s disease: findings from the Colombia-Boston (COLBOS) biomarker study
Additional file 4 of Longitudinal amyloid and tau accumulation in autosomal dominant Alzheimer’s disease: findings from the Colombia-Boston (COLBOS) biomarker study
Additional file 5 of Longitudinal amyloid and tau accumulation in autosomal dominant Alzheimer’s disease: findings from the Colombia-Boston (COLBOS) biomarker study
Additional file 5 of Longitudinal amyloid and tau accumulation in autosomal dominant Alzheimer’s disease: findings from the Colombia-Boston (COLBOS) biomarker study
Additional file 1 of Characterization of Alzheimer’s tau biomarker discordance using plasma, CSF, and PET
Additional file 1 of Characterization of Alzheimer’s tau biomarker discordance using plasma, CSF, and PET
Additional file 1 of Age, vascular disease, and Alzheimer’s disease pathologies in amyloid negative elderly adults
Additional file 1 of Age, vascular disease, and Alzheimer’s disease pathologies in amyloid negative elderly adults
Additional file 1 of The relationship of insulin resistance and diabetes to tau PET SUVR in middle-aged to older adults
Additional file 1 of The relationship of insulin resistance and diabetes to tau PET SUVR in middle-aged to older adults
Additional file 1 of Head-to-head comparison between plasma p-tau217 and flortaucipir-PET in amyloid-positive patients with cognitive impairment
Additional file 1 of Head-to-head comparison between plasma p-tau217 and flortaucipir-PET in amyloid-positive patients with cognitive impairment