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A small sustained increase in NOD1 abundance promotes ligand-independent inflammatory and oncogene transcriptional responses

Science Signaling(2020)10.1126/scisignal.aba3244Source: DataRank Database

A small sustained increase in NOD1 abundance promotes ligand-independent inflammatory and oncogene transcriptional responses is a research paper published in Science Signaling (2020). On theSindex it has a DataRank of 0. It has been cited 8 times.

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Scored on demand from live citation data

Open Access8 citations

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Enrichment:Funding (4 grants)Impact (FWCI 0.48)OA: greenTopics (43)IDs (PubMed, PMC, OpenAlex)Annotations (2)SDGs (4)

FAIR Checklist

Context only (not used in score)

Findable (1/2)

Has DOI

Accessible (1/2)

Open Access

Interoperable (0/2)

Reusable (0/3)

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Authors (10)

Ajay Suresh Akhade ORCID,Bhaskar Dutta ORCID,Carolyn Hutcheon ORCID,Nicolas Lounsbury ORCID,Clifford Rostomily ORCID

Live enrichment

PubMed:33293463 PMC:PMC7853416 OpenAlex:https://openalex.org/W3110909236

National Institutes of Health

Grant: R21-AI138258

NIAID NIH HHS

Grant: R21 AI138258

National Institutes of Health

Grant: 5R21AI138258-02

Can a modest change in regulation of NOD1 expression have a major impact on inflammation and gastric cancer?

Division of Intramural Research, National Institute of Allergy and Infectious Diseases

FWCI

0.48

Citation Percentile

0.6%

Citation Trend

2021

2022

2023

2024

green

repositoryhttps://www.ncbi.nlm.nih.gov/pmc/articles/7853416

GREENhttps://www.ncbi.nlm.nih.gov/pmc/articles/PMC7853416

repositoryhttps://www.ncbi.nlm.nih.gov/pmc/articles/7853416

publisherhttps://syndication.highwire.org/content/doi/10.1126/scisignal.aba3244

publisherhttps://www.science.org/doi/pdf/10.1126/scisignal.aba3244

Fields of Study

Immune Response and InflammationNeutrophil, Myeloperoxidase and Oxidative MechanismsImmune Cell Function and InteractionMedicineChemistryBiology0301 basic medicine0303 health sciences03 medical and health sciences

MeSH Terms

THP-1 CellsGene Expression RegulationHumansInflammationMonocytesTranscription, GeneticSignal TransductionOncogene ProteinsNod1 Signaling Adaptor Protein

Keywords

Abundance (ecology)OncogeneBiologyNOD1InflammationPathogenLigand (biochemistry)Cell biologyImmunologyCancer researchReceptorEcologyCancerGeneticsImmune systemInnate immune systemCell cycleOncogene ProteinsTranscription, GeneticTHP-1 CellsMonocytesGene Expression RegulationNod1 Signaling Adaptor ProteinHumansSignal Transduction

Sustainable Development Goals

SDG 3: 3. Good healthSDG 10: 10. No inequalitySDG 12: 12. Responsible consumptionSDG 0: Zero hunger

A small sustained increase in NOD1 abundance promotes ligand-independent inflammatory and oncogene transcriptional responses

FAIR Checklist

Authors (10)

Funding (4 grants)

Impact

Open Access

Topics

Annotations (Europe PMC)