Lung and gut microbiota are altered by hyperoxia and contribute to oxygen-induced lung injury in mice is a research paper published in Science Translational Medicine (2020). On theSindex it has a DataRank of 0.774. It has been cited 173 times.
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Base Score Contribution
0.774
From this paper's citation signal
Citation Network Contribution
0
Citation network not refreshed for this result
This paper's DataRank is currently driven only by its base citation score. Citation network data was not refreshed for this result.
Learn more about DataRank methodology →National Institutes of Health
Grant: T32HL007517
National Institutes of Health
Grant: K23HL109149
National Institutes of Health
Grant: K23HL130641
National Institutes of Health
Grant: R21AI137669
National Institutes of Health
Grant: R01HL121774
National Institutes of Health
Grant: K01HL136687
National Institutes of Health
Grant: R01HL144599
National Institutes of Health
Grant: R01GM099549
National Institutes of Health
Grant: R01HL140572
NHLBI NIH HHS
Grant: L30 HL120241
NIGMS NIH HHS
Grant: T32 GM007863
National Institutes of Health
Grant: 1R01GM099549-01A1
Impacts of Molecular Oxygen on the Structure and Function of the Intestinal Micro
National Institutes of Health
Grant: 5R21AI137669-02
Rapid pathogen identification in pneumonia using real-time metagenomics and ultrasensitive PCR
National Institutes of Health
Grant: 5K23HL130641-05
The Role of the Microbiome in the Acute Respiratory Distress Syndrome
National Institutes of Health
Grant: 1K01HL136687-01
Data-Driven Identification of the Acute Respiratory Distress Syndrome
National Institutes of Health
Grant: 5K23HL109149-05
Mesenchymal stromal cells and bronchopulmonary dysplasia
National Institutes of Health
Grant: 5R01HL144599-04
The role of the lung microbiome in oxygen-induced lung injury
National Institutes of Health
Grant: 5R01HL121774-02
Functional Analysis of the Pulmonary Microbiome during COPD
National Institutes of Health
Grant: 5T32HL007517-05
LUNG IMMUNOPATHOLOGY
National Institutes of Health
Grant: 5R01HL140572-05
Early life hyperoxic exposure, lung innate immune responses, bronchopulmonary dysplasia and asthma
FWCI
12.74
Citation Percentile
1.0%
Citation Trend
Fields of Study
MeSH Terms
Keywords
Sustainable Development Goals
Additional file 2 of Whole lung tissue is the preferred sampling method for amplicon-based characterization of murine lung microbiota
Additional file 2 of Whole lung tissue is the preferred sampling method for amplicon-based characterization of murine lung microbiota
Additional file 1 of Trajectories of hypoxemia and pulmonary mechanics of COVID-19 ARDS in the NorthCARDS dataset
Additional file 1 of Trajectories of hypoxemia and pulmonary mechanics of COVID-19 ARDS in the NorthCARDS dataset
Additional file 1 of Effect of invasive mechanical ventilation on the diversity of the pulmonary microbiota
Additional file 1 of Effect of invasive mechanical ventilation on the diversity of the pulmonary microbiota
Additional file 1 of Gut microbiota composition during hospitalization is associated with 60-day mortality after severe COVID-19
Additional file 1 of Gut microbiota composition during hospitalization is associated with 60-day mortality after severe COVID-19
Additional file 8 of Intranasal administration of Lactobacillus johnsonii attenuates hyperoxia-induced lung injury by modulating gut microbiota in neonatal mice
Additional file 8 of Intranasal administration of Lactobacillus johnsonii attenuates hyperoxia-induced lung injury by modulating gut microbiota in neonatal mice
Additional file 6 of Intranasal administration of Lactobacillus johnsonii attenuates hyperoxia-induced lung injury by modulating gut microbiota in neonatal mice
Additional file 4 of Intranasal administration of Lactobacillus johnsonii attenuates hyperoxia-induced lung injury by modulating gut microbiota in neonatal mice
Additional file 7 of Intranasal administration of Lactobacillus johnsonii attenuates hyperoxia-induced lung injury by modulating gut microbiota in neonatal mice
Additional file 7 of Intranasal administration of Lactobacillus johnsonii attenuates hyperoxia-induced lung injury by modulating gut microbiota in neonatal mice
Additional file 3 of Intranasal administration of Lactobacillus johnsonii attenuates hyperoxia-induced lung injury by modulating gut microbiota in neonatal mice
Additional file 5 of Intranasal administration of Lactobacillus johnsonii attenuates hyperoxia-induced lung injury by modulating gut microbiota in neonatal mice
Additional file 5 of Intranasal administration of Lactobacillus johnsonii attenuates hyperoxia-induced lung injury by modulating gut microbiota in neonatal mice
Additional file 6 of Intranasal administration of Lactobacillus johnsonii attenuates hyperoxia-induced lung injury by modulating gut microbiota in neonatal mice
Additional file 4 of Intranasal administration of Lactobacillus johnsonii attenuates hyperoxia-induced lung injury by modulating gut microbiota in neonatal mice
Additional file 1 of Intranasal administration of Lactobacillus johnsonii attenuates hyperoxia-induced lung injury by modulating gut microbiota in neonatal mice