APOE genotype regulates pathology and disease progression in synucleinopathy is a research paper published in Science Translational Medicine (2020). On theSindex it has a DataRank of 0.785. It has been cited 186 times.
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Base Score Contribution
0.785
From this paper's citation signal
Citation Network Contribution
0
Citation network not refreshed for this result
This paper's DataRank is currently driven only by its base citation score. Citation network data was not refreshed for this result.
Learn more about DataRank methodology →National Institutes of Health
Grant: K08NS101118
National Institutes of Health
Grant: R01AG044546
National Institutes of Health
Grant: RF1AG053303
National Institutes of Health
Grant: RF1AG058501
National Institutes of Health
Grant: U01AG052411
National Institutes of Health
Grant: U01AG058922
National Institutes of Health
Grant: NS097799
National Institutes of Health
Grant: R01NS090934
National Institutes of Health
Grant: R01AG047644
National Institutes of Health
Grant: NS075321
NINDS NIH HHS
Grant: R01 NS075321
NINDS NIH HHS
Grant: RF1 NS075321
NINDS NIH HHS
Grant: R01 NS097799
Mary E. Groff Surgical and Medical Research and Education Charitable Trust
The Greater St. Louis Chapter of the APDA
American Academy of Neurology
Barnes-Jewish Hospital Foundation
American Brain Foundation
BrightFocus Foundation
The American Parkinson Disease Association
the JPB Foundation
Dobbins Family Fund
The Riney Foundation
FWCI
8.72
Citation Percentile
1.0%
Citation Trend
Fields of Study
MeSH Terms
Keywords
Additional file 1 of Functional genomic analyses uncover APOE-mediated regulation of brain and cerebrospinal fluid beta-amyloid levels in Parkinson disease
Additional file 1 of Functional genomic analyses uncover APOE-mediated regulation of brain and cerebrospinal fluid beta-amyloid levels in Parkinson disease
Additional file 1 of Plasma apolipoprotein E levels in longitudinally followed patients with mild cognitive impairment and Alzheimer’s disease
Additional file 1 of Plasma apolipoprotein E levels in longitudinally followed patients with mild cognitive impairment and Alzheimer’s disease
Additional file 1 of LRP1 is a neuronal receptor for α-synuclein uptake and spread
Additional file 1 of LRP1 is a neuronal receptor for α-synuclein uptake and spread
Additional file 1 of The presence and co-incidence of geriatric syndromes in older patients with mild-moderate Lewy body dementia
Additional file 1 of The presence and co-incidence of geriatric syndromes in older patients with mild-moderate Lewy body dementia
Additional file 1 of SNCA genetic lowering reveals differential cognitive function of alpha-synuclein dependent on sex
Additional file 1 of SNCA genetic lowering reveals differential cognitive function of alpha-synuclein dependent on sex
Additional file 1 of Carboxyl truncation of α-synuclein occurs early and is influenced by human APOE genotype in transgenic mouse models of α-synuclein pathogenesis
Additional file 1 of Carboxyl truncation of α-synuclein occurs early and is influenced by human APOE genotype in transgenic mouse models of α-synuclein pathogenesis
Additional file 1 of APOE-ε4 and BIN1 increase risk of Alzheimer’s disease pathology but not specifically of Lewy body pathology
Additional file 1 of APOE-ε4 and BIN1 increase risk of Alzheimer’s disease pathology but not specifically of Lewy body pathology
Additional file 1 of Association between APOE-ε4 allele and cognitive function is mediated by Alzheimer’s disease pathology: a population-based autopsy study in an admixed sample
Additional file 1 of Association between APOE-ε4 allele and cognitive function is mediated by Alzheimer’s disease pathology: a population-based autopsy study in an admixed sample
Additional file 2 of SNCA genetic lowering reveals differential cognitive function of alpha-synuclein dependent on sex
Additional file 2 of SNCA genetic lowering reveals differential cognitive function of alpha-synuclein dependent on sex