Cis P-tau underlies vascular contribution to cognitive impairment and dementia and can be effectively targeted by immunotherapy in mice is a research paper published in Science Translational Medicine (2021). On theSindex it has a DataRank of 0. It has been cited 67 times.
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National Institutes of Health
Grant: R01AG055559
National Institutes of Health
Grant: U01NS096835
National Institutes of Health
Grant: 2T32AG000222-26
Training in the Molecular Biology of Neurodegeneration and Alzheimer's Disease
National Institutes of Health
Grant: NIA P30 AG013854
National Institutes of Health
Grant: R01AG046319
University of Kentucky
Grant: MCDN-15-368711
Alzheimer Association
Grant: DVT-14-322623
NIA NIH HHS
Grant: T32 AG000222
NCI NIH HHS
Grant: R35 CA197697
NIA NIH HHS
Grant: P30 AG013854
NHLBI NIH HHS
Grant: P01 HL131477
National Institutes of Health
Grant: 1U01NS096835-01A1
Development of Therapeutic Antibody for Traumatic Brain Injury
National Institutes of Health
Grant: 5P30AG013854-05
CORE--CLINICAL
National Institutes of Health
Grant: 5R01AG055559-02
Role of Tau Conformations in Vascular Contribution to Cognitive Impairment and Dementia
National Institutes of Health
Grant: 5R01AG046319-03
Drug Discovery against the Early, Secreted and Toxic Tau in Alzheimer's Disease
Alzheimer Nadační Fond
Edward N. and Della L. Thome Memorial Foundation
FWCI
4.78
Citation Percentile
1.0%
Citation Trend
Fields of Study
MeSH Terms
Keywords
Sustainable Development Goals
Additional file 3 of Long-term normalization of calcineurin activity in model mice rescues Pin1 and attenuates Alzheimer’s phenotypes without blocking peripheral T cell IL-2 response
Additional file 2 of Long-term normalization of calcineurin activity in model mice rescues Pin1 and attenuates Alzheimer’s phenotypes without blocking peripheral T cell IL-2 response
Additional file 4 of Long-term normalization of calcineurin activity in model mice rescues Pin1 and attenuates Alzheimer’s phenotypes without blocking peripheral T cell IL-2 response
Additional file 4 of Long-term normalization of calcineurin activity in model mice rescues Pin1 and attenuates Alzheimer’s phenotypes without blocking peripheral T cell IL-2 response
Additional file 3 of Long-term normalization of calcineurin activity in model mice rescues Pin1 and attenuates Alzheimer’s phenotypes without blocking peripheral T cell IL-2 response
Additional file 2 of Long-term normalization of calcineurin activity in model mice rescues Pin1 and attenuates Alzheimer’s phenotypes without blocking peripheral T cell IL-2 response
Additional file 1 of Long-term normalization of calcineurin activity in model mice rescues Pin1 and attenuates Alzheimer’s phenotypes without blocking peripheral T cell IL-2 response
Additional file 1 of Long-term normalization of calcineurin activity in model mice rescues Pin1 and attenuates Alzheimer’s phenotypes without blocking peripheral T cell IL-2 response