Small-molecule targeted therapies induce dependence on DNA double-strand break repair in residual tumor cells is a research paper published in Science Translational Medicine (2022). On theSindex it has a DataRank of 0. It has been cited 45 times.
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NCI NIH HHS
Grant: F30 CA220847
NIGMS NIH HHS
Grant: T32 GM007171
NIGMS NIH HHS
Grant: T32 GM145449
NCI NIH HHS
Grant: P30 CA014236
NCI NIH HHS
Grant: F32 CA206234
NCI NIH HHS
Grant: R01 CA211052
NCI NIH HHS
Grant: U54 CA224081
NCI NIH HHS
Grant: R01 CA207083
NCI NIH HHS
Grant: R01 CA231300
NCI NIH HHS
Grant: R01 CA238236
NCI NIH HHS
Grant: P30 CA008748
NCI NIH HHS
Grant: R01 CA169338
NCI NIH HHS
Grant: R01 CA204302
NHGRI NIH HHS
Grant: RM1 HG009490
NCI NIH HHS
Grant: U01 CA217882
National Institutes of Health
Grant: 5R01CA207083-03
Selectively targeting apoptosis in PIK3CA mutant colorectal cancers
National Institutes of Health
Grant: 1U01CA217882-01
The Cancer Target Discovery and Development Network at UCSF
National Institutes of Health
Grant: 1F30CA220847-01
Codon bias imposes a targetable limitation on KRAS-driven therapeutic resistance
National Institutes of Health
Grant: 5R01CA169338-02
Rational Combined Inhibition of NF-kB and EGFR to Optimize Lung Cancer Treatment
National Institutes of Health
Grant: 5T32GM007171-17
MEDICAL SCIENTIST
National Institutes of Health
Grant: 1R01CA204302-01A1
Characterization of YAP as a rational companion target in lung cancer
National Institutes of Health
Grant: 5R01CA211052-03
Optimizing biologically-based rational polytherapy in ALK+ lung cancer
National Institutes of Health
Grant: 1F32CA206234-01
Exploring BCL-XL addiction in pancreatic ductal adenocarcinoma
National Institutes of Health
Grant: 5R01CA231300-03
(PQ7) Defining a new mode of RAS signaling in cancer from cytoplasmic protein granules
National Institutes of Health
Grant: 3U54CA224081-01S1
Bay Area Team Against Resistance
FWCI
3.14
Citation Percentile
0.9%
Influential Citations
1
Citation Trend
Fields of Study
MeSH Terms
Keywords
Sustainable Development Goals
Additional file 1 of Dual inhibition of SUMOylation and MEK conquers MYC-expressing KRAS-mutant cancers by accumulating DNA damage
Additional file 1 of Dual inhibition of SUMOylation and MEK conquers MYC-expressing KRAS-mutant cancers by accumulating DNA damage
Additional file 2 of Dual inhibition of SUMOylation and MEK conquers MYC-expressing KRAS-mutant cancers by accumulating DNA damage
Additional file 2 of Dual inhibition of SUMOylation and MEK conquers MYC-expressing KRAS-mutant cancers by accumulating DNA damage