APOE immunotherapy reduces cerebral amyloid angiopathy and amyloid plaques while improving cerebrovascular function is a research paper published in Science Translational Medicine (2021). On theSindex it has a DataRank of 0. It has been cited 198 times.
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National Institute on Aging
Grant: AG062027
National Institute on Aging
Grant: AG047644
National Institute on Aging
Grant: 2P30AG019610
NIH Office of the Director
Grant: OD021629
National Institute of General Medical Sciences
Grant: 5T32GM008151
National Institute of Neurological Disorders and Stroke
Grant: NS103276
NINDS NIH HHS
Grant: RF1 NS103276
NIA NIH HHS
Grant: P30 AG072980
NIA NIH HHS
Grant: P30 AG019610
NIA NIH HHS
Grant: R01 AG047644
NIH HHS
Grant: S10 OD021629
NIA NIH HHS
Grant: RF1 AG047644
NINDS NIH HHS
Grant: RF1 NS090934
NIA NIH HHS
Grant: F31 AG062027
NIGMS NIH HHS
Grant: T32 GM008151
National Institutes of Health
Grant: 5T32GM008151-19
SYSTEMS AND MOLECULAR NEUROBIOLOGY
National Institutes of Health
Grant: 1RF1NS103276-01A1
IMPACT AND MECHANISMS OF VASCULAR OXIDATIVE STRESS ON CAA PATHOGENESIS AND CAA-RELATED DEMENTIA
National Institutes of Health
Grant: 2P30AG019610-11
Education and Information Transfer Core
JPB Foundation
Fields of Study
MeSH Terms
Keywords
Sustainable Development Goals
Additional file 1 of Murine roseolovirus does not accelerate amyloid-β pathology and human roseoloviruses are not over-represented in Alzheimer disease brains
Additional file 1 of Murine roseolovirus does not accelerate amyloid-β pathology and human roseoloviruses are not over-represented in Alzheimer disease brains
Additional file 2 of Murine roseolovirus does not accelerate amyloid-β pathology and human roseoloviruses are not over-represented in Alzheimer disease brains
Additional file 2 of Murine roseolovirus does not accelerate amyloid-β pathology and human roseoloviruses are not over-represented in Alzheimer disease brains
Additional file 3 of Murine roseolovirus does not accelerate amyloid-β pathology and human roseoloviruses are not over-represented in Alzheimer disease brains
Additional file 3 of Murine roseolovirus does not accelerate amyloid-β pathology and human roseoloviruses are not over-represented in Alzheimer disease brains
Additional file 1 of Astrocytic APOE4 removal confers cerebrovascular protection despite increased cerebral amyloid angiopathy
Additional file 1 of Astrocytic APOE4 removal confers cerebrovascular protection despite increased cerebral amyloid angiopathy
Additional file 2 of Astrocytic APOE4 removal confers cerebrovascular protection despite increased cerebral amyloid angiopathy
Additional file 2 of Astrocytic APOE4 removal confers cerebrovascular protection despite increased cerebral amyloid angiopathy
Additional file 3 of Astrocytic APOE4 removal confers cerebrovascular protection despite increased cerebral amyloid angiopathy
Additional file 3 of Astrocytic APOE4 removal confers cerebrovascular protection despite increased cerebral amyloid angiopathy
Additional file 4 of Astrocytic APOE4 removal confers cerebrovascular protection despite increased cerebral amyloid angiopathy
Additional file 4 of Astrocytic APOE4 removal confers cerebrovascular protection despite increased cerebral amyloid angiopathy
Additional file 5 of Astrocytic APOE4 removal confers cerebrovascular protection despite increased cerebral amyloid angiopathy
Additional file 5 of Astrocytic APOE4 removal confers cerebrovascular protection despite increased cerebral amyloid angiopathy
Additional file 6 of Astrocytic APOE4 removal confers cerebrovascular protection despite increased cerebral amyloid angiopathy
Additional file 6 of Astrocytic APOE4 removal confers cerebrovascular protection despite increased cerebral amyloid angiopathy
Additional file 1 of Amyloid-β (Aβ) immunotherapy induced microhemorrhages are associated with activated perivascular macrophages and peripheral monocyte recruitment in Alzheimer’s disease mice
Additional file 1 of Amyloid-β (Aβ) immunotherapy induced microhemorrhages are associated with activated perivascular macrophages and peripheral monocyte recruitment in Alzheimer’s disease mice