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HPV18 E7 inhibits LATS1 kinase and activates YAP1 by degrading PTPN14

mBio(2024)10.1128/mbio.01811-24Source: DataRank Database

HPV18 E7 inhibits LATS1 kinase and activates YAP1 by degrading PTPN14 is a research paper published in mBio (2024). On theSindex it has a DataRank of 0. It has been cited 13 times.

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Open Access13 citations

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›Methodology & internals

datarank_citation_only_1hop_v6· scope data_onlyMethodology

Pipeline:MetadataData-paper checkEnrichmentCitation networkScoring

Enrichment:Funding (15 grants)Impact (FWCI 2.93)OA: goldTopics (65)IDs (PubMed, PMC, OpenAlex)SDGs (1)

FAIR Checklist

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Findable (1/2)

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Accessible (1/2)

Open Access

Interoperable (0/2)

Reusable (0/3)

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Authors (3)

Joshua Hatterschide ORCID,Elizabeth White ORCID,William J. Blakely ORCID

Live enrichment

PubMed:39248565 PMC:PMC11481495 OpenAlex:https://openalex.org/W4402364611

American Cancer Society

Grant: 131661-RSG-18-048-01-MPC

HHS | NIH | National Institute of Allergy and Infectious Diseases

Grant: R01 AI148431

HHS | NIH | National Institute of Allergy and Infectious Diseases

Grant: R21 AI176035

HHS | NIH | National Cancer Institute

Grant: T32 CA115299

HHS | NIH | National Institute of Dental and Craniofacial Research

Grant: F32 DE032573

HHS | NIH | National Institute of Allergy and Infectious Diseases

Grant: T32 AI007324

HHS | NIH | National Institute of Dental and Craniofacial Research

Grant: F31 DE030365

NIAMS NIH HHS

Grant: P30 AR069589

National Institutes of Health

Grant: 5F32DE032573-02

Determining the mechanism for YAP1 activation by HPV E7 in oropharyngeal carcinoma

National Institutes of Health

Grant: 5F31DE030365-02

Investigating the mechanism by which PTPN14 degradation by HPV E7 represses epithelial differentiation

National Institutes of Health

Grant: 1R21AI176035-01A1

Establishment of basal epithelial identity by papillomavirus oncoproteins

National Institutes of Health

Grant: 5T32CA115299-13

Training in Tumor Virology

National Institutes of Health

Grant: 2T32AI007324-06A2

TRAINING IN VIROLOGY

National Institutes of Health

Grant: 3R01AI148431-03S1

Human Papillomavirus Manipulation of Epithelial Differentiation

National Institutes of Health

Grant: 5P30AR069589-03

Penn Resource-based Center to Support and Translate Skin DiseasesResearch

FWCI

2.93

Citation Percentile

0.9%

Citation Trend

2025

2026

goldLicense: cc-by

journalhttps://doi.org/10.1128/mbio.01811-24

publisherhttps://doi.org/10.1128/mbio.01811-24

publisherhttps://journals.asm.org/doi/pdf/10.1128/mbio.01811-24

repositoryhttps://pubmed.ncbi.nlm.nih.gov/39248565

repositoryhttps://www.ncbi.nlm.nih.gov/pmc/articles/11481495

Fields of Study

Hippo pathway signaling and YAP/TAZCancer-related Molecular PathwaysGenital Health and Disease0301 basic medicine03 medical and health sciencesHumansYAP-Signaling ProteinsProtein Serine-Threonine KinasesKeratinocytesAdaptor Proteins, Signal TransducingPhosphorylationTranscription FactorsProtein Tyrosine Phosphatases, Non-ReceptorCell DifferentiationHuman papillomavirus 18Oncogene Proteins, ViralSignal TransductionHost-Pathogen InteractionsDNA-Binding Proteins

MeSH Terms

YAP-Signaling ProteinsCell DifferentiationDNA-Binding ProteinsHumansOncogene Proteins, ViralPhosphorylationTranscription FactorsSignal TransductionKeratinocytesProtein Serine-Threonine KinasesAdaptor Proteins, Signal TransducingHuman papillomavirus 18Protein Tyrosine Phosphatases, Non-ReceptorHost-Pathogen Interactions

Keywords

YAP1Hippo signaling pathwayKinaseBiologyCell biologyCancer researchPhosphorylationTranscription factorBiochemistryDifferentiationHuman papillomavirusKeratinocyteTumor suppressorHippoPtpn14KeratinocytesProtein Serine-Threonine KinasesMicrobiologyArticleHumansAdaptor Proteins, Signal TransducingHuman papillomavirus 18YAP-Signaling ProteinsCell DifferentiationOncogene Proteins, ViralProtein Tyrosine Phosphatases, Non-ReceptorQR1-502DNA-Binding ProteinsHost-Pathogen InteractionsResearch ArticleTranscription FactorsSignal Transduction

Sustainable Development Goals

SDG 0: Good health and well-being