Stress Granules Determine the Development of Obesity-Associated Pancreatic Cancer is a research paper published in Cancer Discovery (2022). On theSindex it has a DataRank of 0. It has been cited 88 times.
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NIH NCI
Grant: R37CA230645
German Research Foundation
Grant: Ga 1818/2-3
German Research Foundation
Grant: SFB1292
National Cancer Center
Grant: P30 CA056036
NCI NIH HHS
Grant: R01 CA160495
Deutsche Forschungsgemeinschaft
Grant: unidentified
unidentified
National Institutes of Health
Grant: 5R37CA230645-05
Stress-adaptation in obesity-associated pancreatic cancer
National Institutes of Health
Grant: 3P30CA056036-22S1
Strengthening Community Engagement in Colorectal Cancer Screening and Clinical Trial Enrollment Through a Community Health Educator
National Institutes of Health
Grant: 5R01CA160495-08
FOXD3 up-regulation and ERBB3 signaling as an adaptive response to BRAF inhibitors in melanoma
FWCI
5.81
Citation Percentile
1.0%
Citation Trend
Fields of Study
MeSH Terms
Keywords
Sustainable Development Goals
Additional file 1 of SUMOylation modulates eIF5A activities in both yeast and pancreatic ductal adenocarcinoma cells
Additional file 1 of SUMOylation modulates eIF5A activities in both yeast and pancreatic ductal adenocarcinoma cells
Additional file 7 of LIPH contributes to glycolytic phenotype in pancreatic ductal adenocarcinoma by activating LPA/LPAR axis and maintaining ALDOA stability
Additional file 7 of LIPH contributes to glycolytic phenotype in pancreatic ductal adenocarcinoma by activating LPA/LPAR axis and maintaining ALDOA stability
Additional file 8 of LIPH contributes to glycolytic phenotype in pancreatic ductal adenocarcinoma by activating LPA/LPAR axis and maintaining ALDOA stability
Additional file 8 of LIPH contributes to glycolytic phenotype in pancreatic ductal adenocarcinoma by activating LPA/LPAR axis and maintaining ALDOA stability
Additional file 6 of LIPH contributes to glycolytic phenotype in pancreatic ductal adenocarcinoma by activating LPA/LPAR axis and maintaining ALDOA stability
Additional file 6 of LIPH contributes to glycolytic phenotype in pancreatic ductal adenocarcinoma by activating LPA/LPAR axis and maintaining ALDOA stability
Additional file 4 of LIPH contributes to glycolytic phenotype in pancreatic ductal adenocarcinoma by activating LPA/LPAR axis and maintaining ALDOA stability
Additional file 5 of LIPH contributes to glycolytic phenotype in pancreatic ductal adenocarcinoma by activating LPA/LPAR axis and maintaining ALDOA stability
Additional file 4 of LIPH contributes to glycolytic phenotype in pancreatic ductal adenocarcinoma by activating LPA/LPAR axis and maintaining ALDOA stability
Additional file 5 of LIPH contributes to glycolytic phenotype in pancreatic ductal adenocarcinoma by activating LPA/LPAR axis and maintaining ALDOA stability
Additional file 3 of LIPH contributes to glycolytic phenotype in pancreatic ductal adenocarcinoma by activating LPA/LPAR axis and maintaining ALDOA stability
Additional file 2 of LIPH contributes to glycolytic phenotype in pancreatic ductal adenocarcinoma by activating LPA/LPAR axis and maintaining ALDOA stability
Additional file 3 of LIPH contributes to glycolytic phenotype in pancreatic ductal adenocarcinoma by activating LPA/LPAR axis and maintaining ALDOA stability
Additional file 1 of LIPH contributes to glycolytic phenotype in pancreatic ductal adenocarcinoma by activating LPA/LPAR axis and maintaining ALDOA stability
Additional file 2 of LIPH contributes to glycolytic phenotype in pancreatic ductal adenocarcinoma by activating LPA/LPAR axis and maintaining ALDOA stability
Additional file 1 of LIPH contributes to glycolytic phenotype in pancreatic ductal adenocarcinoma by activating LPA/LPAR axis and maintaining ALDOA stability