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High Shear Stress Reduces ERG Causing Endothelial-Mesenchymal Transition and Pulmonary Arterial Hypertension

Arteriosclerosis Thrombosis and Vascular Biology(2024)10.1161/atvbaha.124.321092Source: DataRank Database

High Shear Stress Reduces ERG Causing Endothelial-Mesenchymal Transition and Pulmonary Arterial Hypertension is a research paper published in Arteriosclerosis Thrombosis and Vascular Biology (2024). On theSindex it has a DataRank of 0. It has been cited 19 times.

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Open Access19 citations

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geo refseq

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›Methodology & internals

datarank_citation_only_1hop_v6· scope data_onlyMethodology

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Enrichment:Funding (14 grants)Impact (FWCI 5.73)OA: greenTopics (75)IDs (PubMed, PMC, OpenAlex)Annotations (2)SDGs (1)

FAIR Checklist

Context only (not used in score)

Findable (1/2)

Has DOI

Accessible (1/2)

Open Access

Interoperable (0/2)

Reusable (0/3)

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Authors (24)

Jan-Renier Moonen ORCID,Yoon Hong Chun ORCID,Yannick C. Lee-Yow ORCID,Kenichi Okamura ORCID,Jason M. Szafron ORCID

Live enrichment

PubMed:39723537 PMC:PMC11753934 OpenAlex:https://openalex.org/W4405814507

American Heart Association-American Stroke Association

Grant: 20POST35080009 - SARASA ISOBE

NCRR NIH HHS

Grant: S10 RR026917

NHLBI NIH HHS

Grant: T32 HL129970

NHGRI NIH HHS

Grant: R00 HG009917

NHLBI NIH HHS

Grant: T32 HL098049

NHLBI NIH HHS

Grant: R01 HL138473

NHLBI NIH HHS

Grant: R01 HL152134

NHGRI NIH HHS

Grant: K99 HG009917

NHLBI NIH HHS

Grant: P01 HL108797

NHLBI NIH HHS

Grant: R01 HL074186

NHLBI NIH HHS

Grant: R24 HL123767

NHGRI NIH HHS

Grant: R35 HG011324

NIH HHS

Grant: S10 OD010344

NCRR NIH HHS

Grant: UL1 RR024986

FWCI

5.73

Citation Percentile

1.0%

Citation Trend

2025

2026

green

repositoryhttps://pmc.ncbi.nlm.nih.gov/articles/PMC11753934/

publisherhttps://www.ahajournals.org/doi/full/10.1161/ATVBAHA.124.321092

journalhttps://doi.org/10.1161/atvbaha.124.321092

repositoryhttps://pubmed.ncbi.nlm.nih.gov/39723537

Fields of Study

Pulmonary Hypertension Research and TreatmentsCongenital heart defects researchKruppel-like factors researchAnimalsHumansPulmonary ArteryEndothelial CellsStress, MechanicalDisease Models, AnimalTranscriptional Regulator ERGKruppel-Like Factor 4Cells, CulturedEpithelial-Mesenchymal TransitionVascular RemodelingCadherinsMice, Inbred C57BLMechanotransduction, CellularPulmonary Arterial HypertensionKruppel-Like Transcription FactorsHypertension, PulmonaryMiceMaleArterial PressureArteriovenous Shunt, SurgicalBone Morphogenetic Protein Receptors, Type IISnail Family Transcription FactorsSignal TransductionEndothelial-Mesenchymal TransitionOncogene ProteinsAntigens, CDCadherin 5

MeSH Terms

Transcriptional Regulator ERGSnail Family Transcription FactorsPulmonary Arterial HypertensionKruppel-Like Factor 4Endothelial-Mesenchymal TransitionCadherin 5AnimalsArteriovenous Shunt, SurgicalCells, CulturedDisease Models, AnimalHumansHypertension, PulmonaryMaleMice, Inbred C57BLPulmonary ArteryStress, MechanicalTransfectionSignal TransductionOncogene ProteinsAntigens, CDCadherinsMechanotransduction, CellularEndothelial CellsMiceKruppel-Like Transcription FactorsBone Morphogenetic Protein Receptors, Type IIEpithelial-Mesenchymal TransitionArterial PressureVascular Remodeling

Keywords

KLF2ErgCell biologyChemistryMedicineTranscription factorCancer researchInternal medicineEndocrinologyBiologyShear stressEndothelial CellsPulmonary Arterial HypertensionEndothelial-mesenchymal TransitionAorto-caval Shunt Mice

Sustainable Development Goals

SDG 0: Good health and well-being

High Shear Stress Reduces ERG Causing Endothelial-Mesenchymal Transition and Pulmonary Arterial Hypertension

FAIR Checklist

Authors (24)

Funding (14 grants)

Impact

Open Access

Topics

Annotations (Europe PMC)