The Calcineurin-TFEB-p62 Pathway Mediates the Activation of Cardiac Macroautophagy by Proteasomal Malfunction is a research paper published in Circulation Research (2020). On theSindex it has a DataRank of 0. It has been cited 137 times.
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HHS | NIH | National Heart, Lung, and Blood Institute
Grant: HL072166
HHS | NIH | National Heart, Lung, and Blood Institute
Grant: HL085629
HHS | NIH | National Heart, Lung, and Blood Institute
Grant: HL131667
HHS | NIH | National Heart, Lung, and Blood Institute
Grant: HL124248
American Heart Association
Grant: 19TPA34880050
NHLBI NIH HHS
Grant: R01 HL131667
NHLBI NIH HHS
Grant: R01 HL124248
NHLBI NIH HHS
Grant: R01 HL085629
NHLBI NIH HHS
Grant: R01 HL146807
NHLBI NIH HHS
Grant: R01 HL072166
FWCI
11.55
Citation Percentile
1.0%
Citation Trend
Fields of Study
MeSH Terms
Keywords
Sustainable Development Goals
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Additional file 2 of Substantia nigra Smad3 signaling deficiency: relevance to aging and Parkinson’s disease and roles of microglia, proinflammatory factors, and MAPK
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Additional file 7 of Targeted degradation of ⍺-synuclein aggregates in Parkinson’s disease using the AUTOTAC technology
Additional file 7 of Targeted degradation of ⍺-synuclein aggregates in Parkinson’s disease using the AUTOTAC technology
Additional file 2 of Targeted degradation of ⍺-synuclein aggregates in Parkinson’s disease using the AUTOTAC technology
Additional file 4 of Targeted degradation of ⍺-synuclein aggregates in Parkinson’s disease using the AUTOTAC technology
Additional file 1 of Targeted degradation of ⍺-synuclein aggregates in Parkinson’s disease using the AUTOTAC technology
Additional file 3 of Targeted degradation of ⍺-synuclein aggregates in Parkinson’s disease using the AUTOTAC technology
Additional file 2 of Targeted degradation of ⍺-synuclein aggregates in Parkinson’s disease using the AUTOTAC technology
Additional file 6 of Targeted degradation of ⍺-synuclein aggregates in Parkinson’s disease using the AUTOTAC technology