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Sickle Trait and Alpha Thalassemia Increase NOS-Dependent Vasodilation of Human Arteries Through Disruption of Endothelial Hemoglobin-eNOS Interactions

Circulation(2024)10.1161/circulationaha.123.066003Source: DataRank Database

Sickle Trait and Alpha Thalassemia Increase NOS-Dependent Vasodilation of Human Arteries Through Disruption of Endothelial Hemoglobin-eNOS Interactions is a research paper published in Circulation (2024). On theSindex it has a DataRank of 0. It has been cited 4 times.

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Enrichment:Funding (3 grants)OA: hybridTopics (34)IDs (PubMed, PMC)Annotations (3)

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Authors (27)

A. Parker Ruhl ORCID,Xianke Zeng,Phillip Cruz ORCID,Sergio A. Hassan ORCID,Olena Kamenyeva ORCID

Live enrichment

PubMed:39633569 PMC:PMC11670920

Intramural NIH HHS

Grant: ZIA AI001150

National Institutes of Health

Grant: 5P41RR001081-29

FUNCTIONAL PROMISCUITY IN O-SUCCINYLBENZOATE SYNTHASE

National Institutes of Health

Grant: 1ZIAAI001150-04

Nitric Oxide and Endothelial Function in Patients with Malaria

hybridLicense: CC BY NC ND

publisherhttps://www.ahajournals.org/doi/full/10.1161/CIRCULATIONAHA.123.066003

https://doi.org/10.1161/circulationaha.123.066003

https://pubmed.ncbi.nlm.nih.gov/39633569

http://dx.doi.org/10.1161/CIRCULATIONAHA.123.066003

Fields of Study

0301 basic medicine0303 health sciences03 medical and health sciences

MeSH Terms

ArteriesEndothelium, VascularHumansSickle Cell Traitalpha-ThalassemiaNitric OxideHemoglobinsVasodilationAdultMiddle AgedFemaleMaleNitric Oxide Synthase Type IIIalpha-Globinsbeta-Globins

Keywords

Nitric oxideHemoglobinsSickle Cell TraitNitric Oxide Synthase Type IiiAlpha-thalassemiaBeta-globinsAlpha-globinsMaleAdultArteriesMiddle AgedVasodilationOriginal Research ArticlesHumansFemaleEndothelium, Vascular