An inducible Cre mouse for studying roles of the RPE in retinal physiology and disease is a research paper published in JCI Insight (2021). On theSindex it has a DataRank of 0. It has been cited 36 times.
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Dept of Veterans Affairs
Grant: I01BX004939,IK6BX005233,15F-RCS-001
National Institutes of Health
Grant: R24EY027283,R01EY009339,P30EY025585,R01EY029341,F30EY029136,1UL1 TR001412
NEI NIH HHS
Grant: P30 EY025585
NEI NIH HHS
Grant: R24 EY027283
NIGMS NIH HHS
Grant: T32 GM007250
NEI NIH HHS
Grant: F30 EY029136
BLRD VA
Grant: I01 BX004939
NIGMS NIH HHS
Grant: T32 GM008803
BLRD VA
Grant: IK6 BX005233
NEI NIH HHS
Grant: R01 EY009339
NEI NIH HHS
Grant: R01 EY029341
NCATS NIH HHS
Grant: UL1 TR001412
National Institutes of Health
Grant: 5R01EY009339-33
Retinoids in Vision
National Institutes of Health
Grant: 5R01EY029341-04
Development and characterization of mouse models of RP59 DHDDS deficiency
National Institutes of Health
Grant: 5T32GM008803-17
Pre-doctoral Training Program in Molecular Therapeutics
National Institutes of Health
Grant: 5F30EY029136-02
CRISPR/Cas9-Mediated Correction of Abca4 Mutations in a Stargardt Mouse Model
National Institutes of Health
Grant: 1I01BX004939-01
Modulation of retinoid reactivity and pathological signaling in retinal therapeutics
National Institutes of Health
Grant: 3UL1TR001412-08S3
Buffalo Clinical and Translational Research Center
National Institutes of Health
Grant: 5P30EY025585-04
NEI CENTER CORE GRANT FOR VISION RESEARCH
National Institutes of Health
Grant: 5IK6BX005233-03
BLR&D Research Career Scientist Award
National Institutes of Health
Grant: 5R24EY027283-05
Use of systems pharmacology to prevent rod and cone photoreceptor degeneration
FWCI
2.29
Citation Percentile
0.9%
Citation Trend
Fields of Study
MeSH Terms
Keywords
Sustainable Development Goals
Additional file 1 of Blue light exposure collapses the inner blood-retinal barrier by accelerating endothelial CLDN5 degradation through the disturbance of GNAZ and the activation of ADAM17
Additional file 1 of Blue light exposure collapses the inner blood-retinal barrier by accelerating endothelial CLDN5 degradation through the disturbance of GNAZ and the activation of ADAM17
Additional file 1 of Targeting miR-181a/b in retinitis pigmentosa: implications for disease progression and therapy
Additional file 1 of Targeting miR-181a/b in retinitis pigmentosa: implications for disease progression and therapy
Additional file 2 of Targeting miR-181a/b in retinitis pigmentosa: implications for disease progression and therapy
Additional file 2 of Targeting miR-181a/b in retinitis pigmentosa: implications for disease progression and therapy
Additional file 3 of Targeting miR-181a/b in retinitis pigmentosa: implications for disease progression and therapy
Additional file 3 of Targeting miR-181a/b in retinitis pigmentosa: implications for disease progression and therapy
Additional file 2 of Blue light exposure collapses the inner blood-retinal barrier by accelerating endothelial CLDN5 degradation through the disturbance of GNAZ and the activation of ADAM17
Additional file 2 of Blue light exposure collapses the inner blood-retinal barrier by accelerating endothelial CLDN5 degradation through the disturbance of GNAZ and the activation of ADAM17