Leukocytes mediate disease pathogenesis in the Ndufs4(KO) mouse model of Leigh syndrome is a research paper published in JCI Insight (2022). On theSindex it has a DataRank of 0. It has been cited 68 times.
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NIH Office of the Director
Grant: NIH/GM R01 133865
Northwest Mitochondrial Research Guild
Grant: n/a
NIH Office of the Director
Grant: T32 GM086270
NIH Office of the Director
Grant: R00 126147
NIGMS NIH HHS
Grant: K99 GM126147
NHLBI NIH HHS
Grant: R00 HL145004
NIGMS NIH HHS
Grant: R01 GM133865
NINDS NIH HHS
Grant: F31 NS120442
NIGMS NIH HHS
Grant: R00 GM126147
National Institutes of Health
Grant: 3T32GM086270-13S1
Anesthesiology & Perioperative Medicine Research Training
National Institutes of Health
Grant: 5R01GM133865-04
The role of ketone metabolism in sequelae resulting from volatile anesthetic exposure.
National Institutes of Health
Grant: 5R00GM126147-04
The role of mTOR in mitochondrial encephalopathy
FWCI
4.74
Citation Percentile
1.0%
Citation Trend
Fields of Study
MeSH Terms
Keywords
Sustainable Development Goals
Additional file 1 of Interleukin-6-elicited chronic neuroinflammation may decrease survival but is not sufficient to drive disease progression in a mouse model of Leigh syndrome
Additional file 1 of Interleukin-6-elicited chronic neuroinflammation may decrease survival but is not sufficient to drive disease progression in a mouse model of Leigh syndrome
Additional file 1 of Inflammatory and interferon gene expression signatures in patients with mitochondrial disease
Additional file 1 of Inflammatory and interferon gene expression signatures in patients with mitochondrial disease
Additional file 2 of Interleukin-6-elicited chronic neuroinflammation may decrease survival but is not sufficient to drive disease progression in a mouse model of Leigh syndrome
Additional file 2 of Interleukin-6-elicited chronic neuroinflammation may decrease survival but is not sufficient to drive disease progression in a mouse model of Leigh syndrome