COVID-19 severity associates with pulmonary redistribution of CD1c+ DCs and inflammatory transitional and nonclassical monocytes is a research paper published in Journal of Clinical Investigation (2020). On theSindex it has a DataRank of 0.807. It has been cited 216 times.
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Base Score Contribution
0.807
From this paper's citation signal
Citation Network Contribution
0
Citation network not refreshed for this result
This paper's DataRank is currently driven only by its base citation score. Citation network data was not refreshed for this result.
Learn more about DataRank methodology âComunidad de Madrid Talento Program
Grant: 2017-T1/BMD-5396
RamĂłn y Cajal Program
Grant: RYC2018-024374-I
National Institutes of Health
Grant: R21AI140930
MINECO RETOS program
Grant: RTI2018-097485-A-I00
Fondo SUPERA COVID19
Grant: PREDINMUN-COVID
Ministerio de EconomĂa y Competitividad
Grant: SAF2017-82886-R
La Caixa Banking Foundation
Grant: HR17-00016
Fondo de Investigaciones Sanitarias
Grant: PI19/00549
Fondo de Investigaciones Sanitarias
Grant: PI18/01163
National Institutes of Health
Grant: 5R21AI140930-02
Tailored induction of HIV-1 specific immunity through engineered dendritic cell vaccines
FWCI
0.00
Citation Percentile
0.0%
Fields of Study
MeSH Terms
Keywords
Sustainable Development Goals
Additional file 1 of Restoration of dendritic cell homeostasis and Type I/Type III interferon levels in convalescent COVID-19 individuals
Additional file 1 of Restoration of dendritic cell homeostasis and Type I/Type III interferon levels in convalescent COVID-19 individuals
Additional file 2 of Restoration of dendritic cell homeostasis and Type I/Type III interferon levels in convalescent COVID-19 individuals
Additional file 2 of Restoration of dendritic cell homeostasis and Type I/Type III interferon levels in convalescent COVID-19 individuals
Additional file 3 of Restoration of dendritic cell homeostasis and Type I/Type III interferon levels in convalescent COVID-19 individuals
Additional file 3 of Restoration of dendritic cell homeostasis and Type I/Type III interferon levels in convalescent COVID-19 individuals
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Additional file 1 of CD1C is associated with breast cancer prognosis and immune infiltrates
Additional file 10 of Comprehensive DNA methylation profiling of COVID-19 and hepatocellular carcinoma to identify common pathogenesis and potential therapeutic targets
Additional file 10 of Comprehensive DNA methylation profiling of COVID-19 and hepatocellular carcinoma to identify common pathogenesis and potential therapeutic targets
Additional file 11 of Comprehensive DNA methylation profiling of COVID-19 and hepatocellular carcinoma to identify common pathogenesis and potential therapeutic targets
Additional file 11 of Comprehensive DNA methylation profiling of COVID-19 and hepatocellular carcinoma to identify common pathogenesis and potential therapeutic targets
Additional file 1 of Impaired balance between neutrophil extracellular trap formation and degradation by DNases in COVID-19 disease
Additional file 1 of Impaired balance between neutrophil extracellular trap formation and degradation by DNases in COVID-19 disease
Additional file 9 of Comprehensive DNA methylation profiling of COVID-19 and hepatocellular carcinoma to identify common pathogenesis and potential therapeutic targets
Additional file 9 of Comprehensive DNA methylation profiling of COVID-19 and hepatocellular carcinoma to identify common pathogenesis and potential therapeutic targets
Additional file 8 of Comprehensive DNA methylation profiling of COVID-19 and hepatocellular carcinoma to identify common pathogenesis and potential therapeutic targets
Additional file 8 of Comprehensive DNA methylation profiling of COVID-19 and hepatocellular carcinoma to identify common pathogenesis and potential therapeutic targets
Additional file 4 of Comprehensive DNA methylation profiling of COVID-19 and hepatocellular carcinoma to identify common pathogenesis and potential therapeutic targets
Additional file 3 of Comprehensive DNA methylation profiling of COVID-19 and hepatocellular carcinoma to identify common pathogenesis and potential therapeutic targets