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An expanded polyglutamine in ATAXIN1 results in a loss-of-function that exacerbates severity of Multiple Sclerosis in an EAE mouse model

Journal of Neuroinflammation(2025)10.1186/s12974-025-03450-2Source: DataRank Database

An expanded polyglutamine in ATAXIN1 results in a loss-of-function that exacerbates severity of Multiple Sclerosis in an EAE mouse model is a research paper published in Journal of Neuroinflammation (2025). On theSindex it has a DataRank of 0. It has been cited 4 times.

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0DataRank · unranked

Scored on demand from live citation data

Open Access4 citations

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›Methodology & internals

datarank_citation_only_1hop_v6· scope data_onlyMethodology

Pipeline:MetadataData-paper checkEnrichmentCitation networkScoring

Enrichment:Funding (2 grants)Impact (FWCI 1.87)OA: goldTopics (69)IDs (PubMed, PMC, OpenAlex)SDGs (1)

FAIR Checklist

Context only (not used in score)

Findable (1/2)

Has DOI

Accessible (1/2)

Open Access

Interoperable (0/2)

Reusable (0/3)

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Authors (7)

Lisa Duvick ORCID,Praseuth Yang ORCID,Brennon O’Callaghan ORCID,Gavin J. Fuchs,Marija Cvetanović ORCID

Live enrichment

PubMed:40307815 PMC:PMC12044863 OpenAlex:https://openalex.org/W4410019460

NIH

Grant: R35NS127248

National Institutes of Health

Grant: 5R35NS127248-03

Molecular genetics of neurodegenerative pathogenic and protective pathways: The SCA1 perspective

FWCI

1.87

Citation Percentile

0.8%

Citation Trend

2025

2026

goldLicense: cc-by-nc-nd

journalhttps://jneuroinflammation.biomedcentral.com/counter/pdf/10.1186/s12974-025-03450-2

publisherhttps://jneuroinflammation.biomedcentral.com/counter/pdf/10.1186/s12974-025-03450-2

publisherhttps://link.springer.com/content/pdf/10.1186/s12974-025-03450-2.pdf

publisherhttps://link.springer.com/article/10.1186/s12974-025-03450-2/fulltext.html

journalhttps://doi.org/10.1186/s12974-025-03450-2

Fields of Study

Genetic Neurodegenerative DiseasesAmyotrophic Lateral Sclerosis ResearchAutoimmune Neurological Disorders and Treatments03 medical and health sciences0302 clinical medicineAnimalsEncephalomyelitis, Autoimmune, ExperimentalMiceAtaxin-1PeptidesMice, TransgenicDisease Models, AnimalMice, Inbred C57BLFemaleMultiple SclerosisHumansMyelin-Oligodendrocyte GlycoproteinPeptide Fragments

MeSH Terms

Ataxin-1AnimalsDisease Models, AnimalEncephalomyelitis, Autoimmune, ExperimentalFemaleHumansMice, Inbred C57BLMice, TransgenicMultiple SclerosisPeptide FragmentsPeptidesMiceMyelin-Oligodendrocyte Glycoprotein

Keywords

Polyglutamine tractExperimental autoimmune encephalomyelitisMultiple sclerosisImmune systemBiologyNeurodegenerationImmunologyMyelinNeuroinflammationMedicineInflammationNeurosciencePathologyDiseaseHuntington's diseaseCentral nervous systemHuntingtindemyelinationAutoimmuneEAESca1Ataxin1Encephalomyelitis, Autoimmune, ExperimentalResearchMice, TransgenicArticlePeptide FragmentsMice, Inbred C57BLMiceDisease Models, AnimalAnimalsHumansFemaleMyelin-Oligodendrocyte GlycoproteinNeurology. Diseases of the nervous systemRC346-429PeptidesAtaxin-1

Sustainable Development Goals

SDG 0: Good health and well-being