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Functional genomic analyses uncover APOE-mediated regulation of brain and cerebrospinal fluid beta-amyloid levels in Parkinson disease

Acta Neuropathologica Communications(2020)10.1186/s40478-020-01072-8Source: DataRank Database

Functional genomic analyses uncover APOE-mediated regulation of brain and cerebrospinal fluid beta-amyloid levels in Parkinson disease is a research paper published in Acta Neuropathologica Communications (2020). On theSindex it has a DataRank of 0.470. It has been cited 22 times.

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Open Access22 citations

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Enrichment:Funding (36 grants)OA: goldTopics (48)Linked Datasets (6)DataCite Reuse (6)IDs (PubMed, PMC)Annotations (1)SDGs (1)

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Base Score 100%Citation Network 0%

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0.470

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Authors (25)

Jorge A. Bahena,Chengran Yang ORCID,Umber Dube ORCID,Fabiana Farias ORCID,John Budde ORCID

Live enrichment

PubMed:33213513 PMC:PMC7678051

National Institute on Aging

Grant: P01AG003991

National Institute on Aging

Grant: U01AG058922

National Institute on Aging

Grant: R01AG057777

National Institute on Aging

Grant: RF1AG058501

National Institute on Aging

Grant: RF1AG053303

National Institute on Aging

Grant: RF1AG044546

National Institute on Aging

Grant: P50AG005681

National Institute on Aging

Grant: P01AG026276

National Institute of Neurological Disorders and Stroke

Grant: NS075321

National Institute of Neurological Disorders and Stroke

Grant: NS097437

National Institute of Neurological Disorders and Stroke

Grant: R01NS097799

National Institute of Neurological Disorders and Stroke

Grant: R01NS118146

Alzheimer's Association

Grant: NIRG-11-200110

Alzheimer's Association

Grant: BAND-14-338165

Alzheimer's Association

Grant: AARG-16-441560

Alzheimer's Association

Grant: BFG-15-362540

NIA NIH HHS

Grant: R01 AG044546

NINDS NIH HHS

Grant: R01 NS107281

NINDS NIH HHS

Grant: R01 NS075321

NINDS NIH HHS

Grant: RF1 NS075321

NIA NIH HHS

Grant: P30 AG066444

NINDS NIH HHS

Grant: R01 NS097437

NINDS NIH HHS

Grant: K08 NS101118

National Institutes of Health

Grant: 3RF1AG053303-01S1

USING QUANTITATIVE TRAITS TO IDENTIFY NOVEL GENES FOR ALZHEIMERS DISEASE AND OTHER COMPLEX TRAITS

National Institutes of Health

Grant: 5P50AG005681-19

Role of LRP &its ligand tPA in LTP &aging

National Institutes of Health

Grant: 2R01AG058501-02

Genetic Modifiers of TREM2 in Alzheimer's Disease

National Institutes of Health

Grant: 5P01AG026276-07

Antecedent Neuroimaging Biomarkers

National Institutes of Health

Grant: 3R01AG057777-02S1

MENDELIAN RANDOMIZATION FOR UNBIAS BIOMARKER DISCOVERY FOR AD AND OTHER COMPLEX TRAITS

National Institutes of Health

Grant: 5R01AG044546-03

IDENTIFYING RARE VARIANTS THAT INCREASE RISK FOR ALZHEIMER'S DISEASE

National Institutes of Health

Grant: 1R01NS118146-01

Multi-tissue high-throughput proteomic and genomic study in Parkinson's Disease

National Institutes of Health

Grant: 5R01NS075321-08

INVESTIGATIONS OF DEMENTIA IN PARKINSON DISEASE

National Institutes of Health

Grant: 2RF1AG044546-06

Identifying Rare Variants that Increase Risk for Alzheimer's Disease

National Institutes of Health

Grant: 5U01AG058922-05

The Familial Alzheimer Sequencing (FASe) Project

National Institutes of Health

Grant: 5P01AG003991-38

Healthy Aging and Senile Dementia

National Institutes of Health

Grant: 5R01NS097799-04

Protein Aggregation and Neurotransmitter Deficits in Parkinson Disease

National Institutes of Health

Grant: 2R01NS097437-06

Parkinson Disease Clinical Subtypes: Validation, Clinical Utility, and Biological Correlates

goldLicense: cc-by

publisherhttps://actaneurocomms.biomedcentral.com/track/pdf/10.1186/s40478-020-01072-8

publisherhttp://link.springer.com/content/pdf/10.1186/s40478-020-01072-8.pdf

publisherhttp://link.springer.com/article/10.1186/s40478-020-01072-8/fulltext.html

repositoryhttps://www.ncbi.nlm.nih.gov/pmc/articles/7678051

repositoryhttps://digitalcommons.wustl.edu/oa_4/1799

Fields of Study

03 medical and health sciences0302 clinical medicine

MeSH Terms

BrainHumansParkinson DiseasePeptide FragmentsApolipoproteins Etau ProteinsPhosphorylationAgedAged, 80 and overMiddle AgedFemaleMalealpha-SynucleinApolipoprotein E4Genome-Wide Association StudyMendelian Randomization AnalysisAmyloid beta-Peptides

Keywords

apoEParkinson diseaseAmyloid betaalpha-SynucleinGwasMaleApolipoprotein E4610tau Proteins576Apolipoproteins E80 and overMedicine and Health SciencesHumansPhosphorylationRC346-429AgedPsychiatryAged, 80 and overAmyloid beta-PeptidesResearchICTS (Institute of Clinical and Translational Sciences)BrainMendelian Randomization AnalysisMiddle AgedPeptide FragmentsFemaleNeurology. Diseases of the nervous systemGenome-Wide Association Study

Sustainable Development Goals

SDG 3: 3. Good health

Additional file 1 of Functional genomic analyses uncover APOE-mediated regulation of brain and cerebrospinal fluid beta-amyloid levels in Parkinson disease

10.6084/m9.figshare.13265837.v1figshare

Additional file 1 of Functional genomic analyses uncover APOE-mediated regulation of brain and cerebrospinal fluid beta-amyloid levels in Parkinson disease

10.6084/m9.figshare.13265837figshare

Additional file 2 of Functional genomic analyses uncover APOE-mediated regulation of brain and cerebrospinal fluid beta-amyloid levels in Parkinson disease

10.6084/m9.figshare.13265840figshare

Functional genomic analyses uncover APOE-mediated regulation of brain and cerebrospinal fluid beta-amyloid levels in Parkinson disease

10.6084/m9.figshare.c.5214119.v1figshare

Additional file 2 of Functional genomic analyses uncover APOE-mediated regulation of brain and cerebrospinal fluid beta-amyloid levels in Parkinson disease

10.6084/m9.figshare.13265840.v1figshare

Functional genomic analyses uncover APOE-mediated regulation of brain and cerebrospinal fluid beta-amyloid levels in Parkinson disease

10.6084/m9.figshare.c.5214119figshare