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Generation and characterization of monoclonal antibodies against pathologically phosphorylated TDP-43

PLoS ONE(2024)10.1371/journal.pone.0298080Source: DataRank Database

Generation and characterization of monoclonal antibodies against pathologically phosphorylated TDP-43 is a research paper published in PLoS ONE (2024). On theSindex it has a DataRank of 0. It has been cited 6 times.

N/A

0DataRank · unranked

Scored on demand from live citation data

Open Access6 citations

Cite:

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›Methodology & internals

datarank_citation_only_1hop_v6· scope data_onlyMethodology

Pipeline:MetadataData-paper checkEnrichmentCitation networkScoring

Enrichment:Funding (18 grants)Impact (FWCI 1.59)OA: goldTopics (52)IDs (PubMed, OpenAlex)SDGs (1)

FAIR Checklist

Context only (not used in score)

Findable (1/2)

Has DOI

Accessible (1/2)

Open Access

Interoperable (0/2)

Reusable (0/3)

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Authors (14)

Tiffany W. Todd ORCID,Wei Shao ORCID,Caroline J. Jones,Kexin Huang ORCID,Lillian M. Daughrity ORCID

Live enrichment

PubMed:38635657 OpenAlex:https://openalex.org/W4394909923

Foundation for the National Institutes of Health

Grant: R35NS097273

Foundation for the National Institutes of Health

Grant: P01NS084974

Foundation for the National Institutes of Health

Grant: P01NS099114

Foundation for the National Institutes of Health

Grant: U54NS123743

Foundation for the National Institutes of Health

Grant: RF1AG062077

Foundation for the National Institutes of Health

Grant: RF1AG062171

Foundation for the National Institutes of Health

Grant: R01NS117461

Foundation for the National Institutes of Health

Grant: 1R21NS127331

National Institutes of Health

Grant: 1RF1AG062171-01

Nuclear Pore Complex and as a Contributor to Tau Associated Dementia

National Institutes of Health

Grant: 3U54NS123743-04S1

Discovery of novel TDP-43 splicing targets: the Achilles heel for FTD and towards sensitive biomarkers and therapeutic targets

National Institutes of Health

Grant: 7RF1AG062077-02

Novel genetic modifiers of C9orf72 and Tau toxicity

National Institutes of Health

Grant: 5P01NS084974-07

Pathobiology of Neurodegeneration in C9ORF72 repeat expansion

National Institutes of Health

Grant: 1R35NS097273-01

Expanding insights into FTD disease mechanisms

National Institutes of Health

Grant: 1P01NS099114-01

Pathophysiology of genetically defined dementia and neurodegeneration: Defining therapeutic targets and pathways

National Institutes of Health

Grant: 5R01NS117461-04

Poly(ADP-ribose) promotes the condensation and toxicity of C9orf72 arginine-rich dipeptide repeat proteins

National Institutes of Health

Grant: 1R21NS127331-01

Glycogen promotes protein aggregation in ALS and FTD

NIA NIH HHS

Grant: P01 AG019724

NINDS NIH HHS

Grant: R21 NS127331

FWCI

1.59

Citation Percentile

0.8%

Citation Trend

2024

2025

2026

goldLicense: cc-by

journalhttps://doi.org/10.1371/journal.pone.0298080

publisherhttps://doi.org/10.1371/journal.pone.0298080

publisherhttps://dx.plos.org/10.1371/journal.pone.0298080

repositoryhttps://pubmed.ncbi.nlm.nih.gov/38635657

repositoryhttps://www.ncbi.nlm.nih.gov/pmc/articles/11025846

Fields of Study

Amyotrophic Lateral Sclerosis ResearchCholinesterase and Neurodegenerative DiseasesPrion Diseases and Protein Misfolding03 medical and health sciences0302 clinical medicineMiceAnimalsHumansAmyotrophic Lateral SclerosisFrontotemporal DementiaAntibodies, MonoclonalHEK293 CellsDNA-Binding ProteinsTDP-43 ProteinopathiesFrontotemporal Dementia With Motor Neuron Disease

MeSH Terms

Amyotrophic Lateral SclerosisAnimalsAntibodies, MonoclonalDNA-Binding ProteinsHumansMiceTDP-43 ProteinopathiesFrontotemporal DementiaHEK293 Cells

Keywords

Amyotrophic lateral sclerosisMonoclonal antibodyMolecular biologyImmunohistochemistryPhosphorylationBiologyFrontotemporal dementiaAntibodyRecombinant DNAPathologyChemistryDementiaMedicineCell biologyImmunologyBiochemistryDiseaseScienceQRAntibodies, MonoclonalDNA-Binding ProteinsMiceHEK293 CellsTDP-43 ProteinopathiesAnimalsHumansResearch Article

Sustainable Development Goals

SDG 3: 3. Good health

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