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ALS‐linked KIF5A ΔExon27 mutant causes neuronal toxicity through gain‐of‐function

EMBO Reports(2022)10.15252/embr.202154234Source: DataRank Database

ALS‐linked KIF5A ΔExon27 mutant causes neuronal toxicity through gain‐of‐function is a research paper published in EMBO Reports (2022). On theSindex it has a DataRank of 0. It has been cited 70 times.

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0DataRank · unranked

Scored on demand from live citation data

Open Access70 citations

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›Methodology & internals

datarank_citation_only_1hop_v6· scope data_onlyMethodology

Pipeline:MetadataData-paper checkEnrichmentCitation networkScoring

Enrichment:Funding (12 grants)Impact (FWCI 7.04)OA: bronzeTopics (50)IDs (PubMed, OpenAlex)SDGs (2)

FAIR Checklist

Context only (not used in score)

Findable (1/2)

Has DOI

Accessible (1/2)

Open Access

Interoperable (0/2)

Reusable (0/3)

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Authors (14)

Janani Parameswaran ORCID,Lu Rao ORCID,Isabel Loss ORCID,Ganesh Chilukuri ORCID,Rosanna Parlato ORCID

Live enrichment

PubMed:35735139 OpenAlex:https://openalex.org/W4283331346

ALS Association

Grant: 21‐PDF‐585

ALS Association

Grant: 22‐PDF‐605

Deutsche Forschungsgemeinschaft

Grant: 492655220

HHS ¦ NIH ¦ National Institute of Neurological Disorders and Stroke

Grant: R01NS114253

HHS ¦ NIH ¦ National Institute of Neurological Disorders and Stroke

Grant: R01GM098469

HHS ¦ NIH ¦ National Institute of Neurological Disorders and Stroke

Grant: R01NS114636

HHS ¦ NIH ¦ National Institute on Aging

Grant: R01AG068247

National Institutes of Health

Grant: 6R01GM098469-08

Molecular Mechanism of the Cytoplasmic Dynein-Dynactin Motor Complex

National Institutes of Health

Grant: 5R01AG068247-02

Cell-type-specific toxicity of C9orf72 repeat expansions in FTD and ALS

Deutsche Forschungsgemeinschaft

Grant: unidentified

unidentified

National Institutes of Health

Grant: 5R01NS114253-02

Mechanism and Function Of MBNL Mediated mRNA Localization in Neuronal Development and Neurologic Disease

National Institutes of Health

Grant: 5R01NS114636-04

Role of the Kinesin KIF1A in Neurological Disease

FWCI

7.04

Citation Percentile

1.0%

Citation Trend

2022

2023

2024

2025

2026

bronzeLicense: Springer TDM

journalhttps://link.springer.com/content/pdf/10.15252/embr.202154234.pdf

publisherhttps://link.springer.com/content/pdf/10.15252/embr.202154234.pdf

publisherhttps://onlinelibrary.wiley.com/doi/full-xml/10.15252/embr.202154234

publisherhttps://link.springer.com/article/10.15252/embr.202154234/fulltext.html

publisherhttps://www.embopress.org/doi/pdf/10.15252/embr.202154234

Fields of Study

Amyotrophic Lateral Sclerosis ResearchMicrotubule and mitosis dynamicsNeurogenetic and Muscular Disorders Research0301 basic medicine0303 health sciences03 medical and health sciencesAmyotrophic Lateral SclerosisAnimalsDNA, AntisenseDrosophila melanogasterGain of Function MutationHumansKinesinsMotor NeuronsMutationTranscription Factor 7-Like 2 Protein

MeSH Terms

Gain of Function MutationAmyotrophic Lateral SclerosisAnimalsDrosophila melanogasterHumansMotor NeuronsMutationDNA, AntisenseKinesinsTranscription Factor 7-Like 2 Protein

Keywords

Amyotrophic lateral sclerosisMutationMutantBiologyGeneticsDrosophila melanogasterMechanism (biology)Ectopic expressionCell biologyGeneDiseaseMedicineMotor NeuronsKinesinsDNA, AntisenseGain of Function MutationAnimalsHumansTranscription Factor 7-Like 2 ProteinALSKIF5Aaggregationautoinhibitionmicrotubules

Sustainable Development Goals

SDG 3: 3. Good healthSDG 0: Good health and well-being