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Senescence-associated hyper-activation to inflammatory stimuli in vitro

Aging(2021)10.18632/aging.203396Source: DataRank Database

Senescence-associated hyper-activation to inflammatory stimuli in vitro is a research paper published in Aging (2021). On theSindex it has a DataRank of 0. It has been cited 58 times.

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Open Access58 citations

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›Methodology & internals

datarank_citation_only_1hop_v6· scope data_onlyMethodology

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Enrichment:Funding (8 grants)Impact (FWCI 5.48)OA: hybridTopics (69)IDs (PubMed, PMC, OpenAlex)Annotations (2)SDGs (2)

FAIR Checklist

Context only (not used in score)

Findable (1/2)

Has DOI

Accessible (1/2)

Open Access

Interoperable (0/2)

Reusable (0/3)

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Authors (7)

Sahana Manohar‐Sindhu ORCID,Yang Yang ORCID,Yonghan He ORCID,Dmitry O. Traktuev ORCID,Thomas C. Foster ORCID

Live enrichment

PubMed:34375950 PMC:PMC8386536 OpenAlex:https://openalex.org/W3192596710

NIA NIH HHS

Grant: R01 AG052258

NCI NIH HHS

Grant: R01 CA211963

NIA NIH HHS

Grant: P30 AG028740

NIA NIH HHS

Grant: R01 AG037984

National Institutes of Health

Grant: 5R01AG052258-04

Systemic inflammation in regulating the onset and progression of brain aging

National Institutes of Health

Grant: 5P30AG028740-07

RESOURCE CORE 3: BIOSTATISTICS AND DATA MANAGEMENT CORE

National Institutes of Health

Grant: 5R01AG037984-13

Estrogen and cognition over the lifespan

National Institutes of Health

Grant: 7R01CA211963-06

Ionizing radiation induced hematological malignancies

FWCI

5.48

Citation Percentile

1.0%

Citation Trend

2021

2022

2023

2024

2025

2026

hybridLicense: cc-by

journalhttps://www.aging-us.com/article/203396/pdf

publisherhttps://www.aging-us.com/article/203396/pdf

publisherhttps://www.aging-us.com/lookup/doi/10.18632/aging.203396

journalhttps://doi.org/10.18632/aging.203396

repositoryhttps://pubmed.ncbi.nlm.nih.gov/34375950

Fields of Study

Telomeres, Telomerase, and SenescenceNeutrophil, Myeloperoxidase and Oxidative MechanismsNeuroinflammation and Neurodegeneration Mechanisms0301 basic medicine0303 health sciences03 medical and health sciencesCell LineCyclopropanesHumansImidazolesInflammation MediatorsInterleukin-6LipopolysaccharidesNF-kappa BPyridinesQuinoxalinesSenescence-Associated Secretory PhenotypeSenotherapeuticsSignal TransductionTumor Necrosis Factor-alphap38 Mitogen-Activated Protein Kinases

MeSH Terms

Senescence-Associated Secretory PhenotypeSenotherapeuticsCell LineCyclopropanesHumansImidazolesLipopolysaccharidesPyridinesQuinoxalinesTumor Necrosis Factor-alphaSignal TransductionInterleukin-6NF-kappa BInflammation Mediatorsp38 Mitogen-Activated Protein Kinases

Keywords

ChemokineLipopolysaccharideTumor necrosis factor alphaStimulationSenescenceCytokineInflammationp38 mitogen-activated protein kinasesIn vitroMedicineImmunologyBiologySignal transductionCell biologyInternal medicineMAPK/ERK pathwayBiochemistryCellular senescenceSASPCyclopropanesLipopolysaccharidesInterleukin-6PyridinesTumor Necrosis Factor-alphaImidazolesNF-kappa BCell LineSenotherapeuticsQuinoxalinesHumansSenescence-Associated Secretory PhenotypeInflammation MediatorsPriority Research Paper

Sustainable Development Goals

SDG 3: 3. Good healthSDG 0: Good health and well-being