Dlk1 is a negative regulator of emerging hematopoietic stem and progenitor cells is a research paper published in Haematologica (2013). On theSindex it has a DataRank of 1.8. It has been cited 74 times, with 39 citing works in its 1-hop citation network.
Scored on demand from live citation data
Linked data & code
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Base Score Contribution
0.648
From this paper's citation signal
Citation Network Contribution
1.1
From 32 citing papers with measurable signal
Wellcome Trust
Grant: 079249
Blood Cancer UK
Grant: 10015
Medical Research Council
Grant: G0800784B
Wellcome Trust
Grant: 095606
Wellcome Trust
Grant: 095606/Z/11/Z
Medical Research Council
Grant: MR/J001597/1
NIDDK NIH HHS
Grant: R37 DK054077
Medical Research Council
Grant: G1000801h
Medical Research Council
Grant: MC_UU_12009/2
Medical Research Council
Grant: G0800784
FWCI
1.69
Citation Percentile
0.8%
Citation Trend
Fields of Study
MeSH Terms
Keywords
Additional file 2 of Dlk1 maintains adult mice long-term HSCs by activating Notch signaling to restrict mitochondrial metabolism
Additional file 2 of Dlk1 maintains adult mice long-term HSCs by activating Notch signaling to restrict mitochondrial metabolism
Additional file 4 of Dlk1 maintains adult mice long-term HSCs by activating Notch signaling to restrict mitochondrial metabolism
Additional file 4 of Dlk1 maintains adult mice long-term HSCs by activating Notch signaling to restrict mitochondrial metabolism
Additional file 5 of Dlk1 maintains adult mice long-term HSCs by activating Notch signaling to restrict mitochondrial metabolism
Additional file 5 of Dlk1 maintains adult mice long-term HSCs by activating Notch signaling to restrict mitochondrial metabolism
Additional file 6 of Dlk1 maintains adult mice long-term HSCs by activating Notch signaling to restrict mitochondrial metabolism
Additional file 6 of Dlk1 maintains adult mice long-term HSCs by activating Notch signaling to restrict mitochondrial metabolism
Additional file 10 of Dlk1 maintains adult mice long-term HSCs by activating Notch signaling to restrict mitochondrial metabolism
Additional file 10 of Dlk1 maintains adult mice long-term HSCs by activating Notch signaling to restrict mitochondrial metabolism
Additional file 3 of Dlk1 maintains adult mice long-term HSCs by activating Notch signaling to restrict mitochondrial metabolism
Additional file 1 of Dlk1 maintains adult mice long-term HSCs by activating Notch signaling to restrict mitochondrial metabolism
Additional file 7 of Dlk1 maintains adult mice long-term HSCs by activating Notch signaling to restrict mitochondrial metabolism
Additional file 8 of Dlk1 maintains adult mice long-term HSCs by activating Notch signaling to restrict mitochondrial metabolism
Additional file 9 of Dlk1 maintains adult mice long-term HSCs by activating Notch signaling to restrict mitochondrial metabolism
Additional file 9 of Dlk1 maintains adult mice long-term HSCs by activating Notch signaling to restrict mitochondrial metabolism
Additional file 8 of Dlk1 maintains adult mice long-term HSCs by activating Notch signaling to restrict mitochondrial metabolism
Additional file 3 of Dlk1 maintains adult mice long-term HSCs by activating Notch signaling to restrict mitochondrial metabolism
Additional file 1 of Dlk1 maintains adult mice long-term HSCs by activating Notch signaling to restrict mitochondrial metabolism
Additional file 7 of Dlk1 maintains adult mice long-term HSCs by activating Notch signaling to restrict mitochondrial metabolism