Pilot corpus: NIH-funded biomedical datasets. Any DOI can be scored on demand — network effects sharpen as coverage grows.

Hyperglycemic Conditions Promote Rac1-Mediated Serine536 Phosphorylation of p65 Subunit of NFκB (RelA) in Pancreatic Beta Cells

Cellular Physiology and Biochemistry(2022)10.33594/000000557Source: DataRank Database

Hyperglycemic Conditions Promote Rac1-Mediated Serine536 Phosphorylation of p65 Subunit of NFκB (RelA) in Pancreatic Beta Cells is a research paper published in Cellular Physiology and Biochemistry (2022). On theSindex it has a DataRank of 0. It has been cited 2 times.

N/A

0DataRank · unranked

Scored on demand from live citation data

Open Access2 citations

Download PDF

Cite:

DataRank reads this dataset's downstream impact straight off the citation graph — no black box, no proprietary weighting. How is this computed?

›Methodology & internals

datarank_citation_only_1hop_v6· scope data_onlyMethodology

Pipeline:MetadataData-paper checkEnrichmentCitation networkScoring

Enrichment:Funding (8 grants)Impact (FWCI 0.19)OA: goldTopics (45)IDs (PubMed, PMC, OpenAlex)SDGs (2)

FAIR Checklist

Context only (not used in score)

Findable (1/2)

Has DOI

Accessible (1/2)

Open Access

Interoperable (0/2)

Reusable (0/3)

FAIR checklist signals are shown for context only and do not affect DataRank scoring.

Run a calibrated FAIR evaluation for this paper →

We only score data papers we can read in full — never from an abstract alone.

Authors (4)

Suhadinie Gamage,Mirabela Hali ORCID,Noah Gleason ORCID,Anjaneyulu Kowluru ORCID

Live enrichment

PubMed:35981264 PMC:PMC9644397 OpenAlex:https://openalex.org/W4292227719

NIDDK NIH HHS

Grant: P30 DK020572

BLRD VA

Grant: I01 BX004663

BLRD VA

Grant: IK6 BX005383

NIMHD NIH HHS

Grant: P50 MD017351

NEI NIH HHS

Grant: R01 EY022230

NHLBI NIH HHS

Grant: T32 HL120822

National Institutes of Health

Grant: 5T32HL120822-02

Detroit Cardiovascular Training Program

National Institutes of Health

Grant: 5R01EY022230-04

NADPH Oxidase, Mitochondrial Dysfunction and Diabetic Retinopathy

FWCI

0.19

Citation Percentile

0.5%

Citation Trend

2023

2025

goldLicense: cc-by

journalhttps://articles.cellphysiolbiochem.com/Articles/000557/PDF/000557.pdf

publisherhttps://articles.cellphysiolbiochem.com/Articles/000557/PDF/000557.pdf

journalhttps://doi.org/10.33594/000000557

repositoryhttps://pubmed.ncbi.nlm.nih.gov/35981264

repositoryhttps://doaj.org/article/8149b7ad9e804bcd9d48097df0ac3cb4

Fields of Study

NF-κB Signaling PathwaysPancreatic function and diabetesCancer-related Molecular Pathways0301 basic medicine0303 health sciences03 medical and health sciencesGlucoseHumansHyperglycemiaInsulin-Secreting CellsNF-kappa BPhosphorylationTranscription Factor RelArac1 GTP-Binding Protein

MeSH Terms

GlucoseHumansHyperglycemiaPhosphorylationNF-kappa Brac1 GTP-Binding ProteinInsulin-Secreting CellsTranscription Factor RelA

Keywords

PhosphorylationRAC1ChemistryProtein subunitMolecular biologySignal transductionPrenylationCell biologyBiochemistryBiologyEnzymeP65; Rela; Nfκb; Rac1; Metabolic Stress; Islet Β-cell; Small Molecular Inhibitors For Rac1rac1 GTP-Binding ProteinPhysiologyNF-kappa BTranscription Factor RelAQD415-436ArticleGlucoseHyperglycemiaInsulin-Secreting CellsQP1-981Humans

Sustainable Development Goals

SDG 3: 3. Good healthSDG 0: Decent work and economic growth