Pilot corpus: NIH-funded biomedical datasets. Any DOI can be scored on demand — network effects sharpen as coverage grows.

Chronic GPER1 Activation Protects Against Oxidative Stress-Induced Cardiomyoblast Death via Preservation of Mitochondrial Integrity and Deactivation of Mammalian Sterile-20-Like Kinase/Yes-Associated Protein Pathway

Frontiers in Endocrinology(2020)10.3389/fendo.2020.579161Source: DataRank Database

Chronic GPER1 Activation Protects Against Oxidative Stress-Induced Cardiomyoblast Death via Preservation of Mitochondrial Integrity and Deactivation of Mammalian Sterile-20-Like Kinase/Yes-Associated Protein Pathway is a research paper published in Frontiers in Endocrinology (2020). On theSindex it has a DataRank of 0. It has been cited 15 times.

N/A

0DataRank · unranked

Scored on demand from live citation data

Open Access15 citations

Download PDF

Cite:

Linked data & code

cellosaurus

DataRank reads this dataset's downstream impact straight off the citation graph — no black box, no proprietary weighting. How is this computed?

›Methodology & internals

datarank_citation_only_1hop_v6· scope data_onlyMethodology

Pipeline:MetadataData-paper checkEnrichmentCitation networkScoring

Enrichment:Funding (6 grants)OA: goldTopics (42)IDs (PubMed, PMC)Annotations (1)

FAIR Checklist

Context only (not used in score)

Findable (1/2)

Has DOI

Accessible (1/2)

Open Access

Interoperable (0/2)

Reusable (0/3)

FAIR checklist signals are shown for context only and do not affect DataRank scoring.

Run a calibrated FAIR evaluation for this paper →

We only score data papers we can read in full — never from an abstract alone.

Authors (5)

Ngonidzashe B Madungwe ORCID,Nathalie Tombo,Yansheng Feng ORCID,Jean C. Bopassa ORCID,Abdulhafiz Imam Aliagan ORCID

Live enrichment

PubMed:33193095 PMC:PMC7604496

NCATS NIH HHS

Grant: UL1 TR001120

NHLBI NIH HHS

Grant: R01 HL138093

NCI NIH HHS

Grant: P30 CA054174

National Institutes of Health

Grant: 5R01HL138093-03

Degradation of Mitochondrial Inner Membrane Protein Disrupts the Structural Interaction between Mitofilin and Cyclophilin D and Determines the Extent of Ischemia/reperfusion injury

National Institutes of Health

Grant: 5P30CA054174-16

SAN ANTONIO CANCER INSTITUTE

National Institutes of Health

Grant: 3UL1TR001120-02S1

Institute for Integration of Medicine & Science: A Partnership to Improve Health

goldLicense: cc-by

publisherhttps://www.frontiersin.org/articles/10.3389/fendo.2020.579161/pdf

publisherhttps://www.frontiersin.org/articles/10.3389/fendo.2020.579161/full

repositoryhttps://www.ncbi.nlm.nih.gov/pmc/articles/7604496

repositoryhttps://doaj.org/article/ec8651b3c30c42fdba95c2a004716173

Europe_PMChttps://europepmc.org/articles/PMC7604496

Fields of Study

0301 basic medicine03 medical and health sciences

MeSH Terms

MitochondriaMyocytes, CardiacAnimalsRatsRats, Sprague-DawleyReperfusion InjuryIntracellular Signaling Peptides and ProteinsReceptors, G-Protein-CoupledProtective AgentsOxidative StressMaleSerine-Threonine Kinase 3YAP-Signaling ProteinsProtein Serine-Threonine KinasesHippo Kinases

Keywords

Cell cycleMitochondrial DynamicsMitochondrial FunctionMptp OpeningRat CardiomyoblastsMst1/Yap PathwayG Protein-coupled Estrogen Receptor 1 (Gper1)MaleProtein Serine-Threonine KinasesProtective AgentsSerine-Threonine Kinase 3Diseases of the endocrine glands. Clinical endocrinologyReceptors, G-Protein-CoupledRats, Sprague-DawleyEndocrinologyAnimalsMyocytes, CardiacIntracellular Signaling Peptides and ProteinsHippo KinasesYAP-Signaling ProteinsRC648-665MitochondriaRatsOxidative StressReperfusion Injury

Chronic GPER1 Activation Protects Against Oxidative Stress-Induced Cardiomyoblast Death via Preservation of Mitochondrial Integrity and Deactivation of Mammalian Sterile-20-Like Kinase/Yes-Associated Protein Pathway

FAIR Checklist

Authors (5)

Funding (6 grants)

Open Access

Topics

Annotations (Europe PMC)