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CCL3/Macrophage Inflammatory Protein-1α Is Dually Involved in Parasite Persistence and Induction of a TNF- and IFNγ-Enriched Inflammatory Milieu in Trypanosoma cruzi-Induced Chronic Cardiomyopathy

Frontiers in Immunology(2020)10.3389/fimmu.2020.00306Source: DataRank Database

CCL3/Macrophage Inflammatory Protein-1α Is Dually Involved in Parasite Persistence and Induction of a TNF- and IFNγ-Enriched Inflammatory Milieu in Trypanosoma cruzi-Induced Chronic Cardiomyopathy is a research paper published in Frontiers in Immunology (2020). On theSindex it has a DataRank of 0. It has been cited 49 times.

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Open Access49 citations

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Enrichment:Funding (2 grants)Impact (FWCI 4.11)OA: goldTopics (69)IDs (PubMed, PMC, OpenAlex)Annotations (1)SDGs (2)

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Findable (1/2)

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Accessible (1/2)

Open Access

Interoperable (0/2)

Reusable (0/3)

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Authors (9)

Gláucia Vilar-Pereira,Isabela Resende Pereira,Andréa Alice Silva ORCID,Leda Castaño Barrios ORCID,Isalira Peroba Ramos ORCID

Live enrichment

PubMed:32194558 PMC:PMC7063958 OpenAlex:https://openalex.org/W3009337990

NIAID NIH HHS

Grant: R01 AI116577

National Institutes of Health

Grant: 5R01AI116577-05

Granzyme and Granulysin Mediated Death of Trypanosoma cruzi

FWCI

4.11

Citation Percentile

1.0%

Influential Citations

Citation Trend

2019

2020

2021

2022

2023

2024

2025

2026

goldLicense: cc-by

journalhttps://doi.org/10.3389/fimmu.2020.00306

GOLDhttps://doi.org/10.3389/fimmu.2020.00306

publisherhttps://doi.org/10.3389/fimmu.2020.00306

publisherhttps://www.frontiersin.org/article/10.3389/fimmu.2020.00306/full

repositoryhttps://pubmed.ncbi.nlm.nih.gov/32194558

Fields of Study

Trypanosoma species research and implicationsResearch on Leishmaniasis StudiesMedicineBiology0301 basic medicine03 medical and health sciences0303 health sciences

MeSH Terms

AnimalsCells, CulturedChagas CardiomyopathyChemotaxis, LeukocyteElectrocardiographyFemaleInterferon-gammaMaleMice, Inbred C57BLMyocarditisRNA, MessengerSpecific Pathogen-Free OrganismsSpleenStroke VolumeTrypanosoma cruziTumor Necrosis Factor-alphaCytokinesMacrophages, PeritonealMice, KnockoutCD8-Positive T-LymphocytesParasitemiaChemokine CCL5Receptors, ChemokineMiceChemokine CCL3

Keywords

CCL3ChemokineTumor necrosis factor alphaBiologyImmunologyMacrophage inflammatory proteinCCL2InflammationTNFIFNγHeart Diseasecell migrationChagas DiseaseQtc ProlongationMet-rantesChagas CardiomyopathyMaleCD8-Positive T-LymphocytesParasitemiaElectrocardiographyInterferon-gammaMiceAnimalsRNA, MessengerChemokine CCL5Cells, CulturedChemokine CCL3Mice, KnockoutRC581-607Mice, Inbred C57BLChemotaxis, LeukocyteMyocarditisMacrophages, PeritonealCytokinesFemaleReceptors, ChemokineImmunologic diseases. Allergy

Sustainable Development Goals

SDG 3: 3. Good healthSDG 0: Good health and well-being

CCL3/Macrophage Inflammatory Protein-1α Is Dually Involved in Parasite Persistence and Induction of a TNF- and IFNγ-Enriched Inflammatory Milieu in Trypanosoma cruzi-Induced Chronic Cardiomyopathy

FAIR Checklist

Authors (9)

Funding (2 grants)

Impact

Open Access

Topics

Annotations (Europe PMC)