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Targeting Mitochondrial-Derived Reactive Oxygen Species in T Cell-Mediated Autoimmune Diseases

Frontiers in Immunology(2021)10.3389/fimmu.2021.703972Source: DataRank Database

Targeting Mitochondrial-Derived Reactive Oxygen Species in T Cell-Mediated Autoimmune Diseases is a research paper published in Frontiers in Immunology (2021). On theSindex it has a DataRank of 0. It has been cited 158 times.

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0DataRank · unranked

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Open Access158 citations

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›Methodology & internals

datarank_citation_only_1hop_v6· scope data_onlyMethodology

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Enrichment:Funding (3 grants)Impact (FWCI 6.88)OA: goldTopics (35)Linked Datasets (12)DataCite Reuse (12)IDs (PubMed, OpenAlex)SDGs (1)

FAIR Checklist

Context only (not used in score)

Findable (2/2)

Has DOI
Indexed in repositories

Accessible (1/2)

Open Access

Interoperable (2/2)

DataCite relations
Linked datasets

Reusable (0/3)

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Authors (2)

Hubert M. Tse ORCID,Miranda D. Chávez

Live enrichment

PubMed:34276700 OpenAlex:https://openalex.org/W3184561875

National Institutes of Health

Grant: 5R01DK127497-03

Determining the mechanism of IFIH1 disease-associated variants on beta-cell and immune responses in Type 1 diabetes

National Institutes of Health

Grant: 5R01DK099550-04

Redox Regulation of Anti-Viral Responses in Type 1 Diabetes

National Institutes of Health

Grant: 1R56DK126456-01

Beta-cell responses to oxidative stress and Type 1 diabetes

FWCI

6.88

Citation Percentile

1.0%

Citation Trend

2021

2022

2023

2024

2025

2026

goldLicense: cc-by

journalhttps://www.frontiersin.org/articles/10.3389/fimmu.2021.703972/pdf

publisherhttps://www.frontiersin.org/articles/10.3389/fimmu.2021.703972/pdf

publisherhttps://www.frontiersin.org/articles/10.3389/fimmu.2021.703972/full

journalhttps://doi.org/10.3389/fimmu.2021.703972

repositoryhttps://pubmed.ncbi.nlm.nih.gov/34276700

Fields of Study

T-cell and B-cell ImmunologyNeutrophil, Myeloperoxidase and Oxidative MechanismsImmune Cell Function and Interaction0301 basic medicine03 medical and health sciences

MeSH Terms

Autoimmune DiseasesHumansLymphocyte ActivationMitochondriaReactive Oxygen SpeciesT-Lymphocytes, Regulatory

Keywords

T cellFOXP3ImmunologyImmune systemBiologyMitochondrial ROSRegulatory T cellProinflammatory cytokineCytotoxic T cellReactive oxygen speciesCell biologyCancer researchIL-2 receptorInflammationBiochemistryautoimmunityimmunometabolismRC581-607Lymphocyte ActivationT-Lymphocytes, RegulatoryAutoimmune DiseasesMitochondriaHumansImmunologic diseases. Allergy

Sustainable Development Goals

SDG 3: 3. Good health

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10.6084/m9.figshare.24013098.v1figshare

Additional file 1 of A compendium of mitochondrial molecular characteristics provides novel perspectives on the treatment of rheumatoid arthritis patients

10.6084/m9.figshare.24013098figshare

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10.6084/m9.figshare.24934357.v1figshare

Additional file 1 of Monocyte-derived transcriptomes explain the ineffectiveness of abatacept in rheumatoid arthritis

10.6084/m9.figshare.24934357figshare

Additional file 2 of Monocyte-derived transcriptomes explain the ineffectiveness of abatacept in rheumatoid arthritis

10.6084/m9.figshare.24934507figshare

Additional file 2 of Monocyte-derived transcriptomes explain the ineffectiveness of abatacept in rheumatoid arthritis

10.6084/m9.figshare.24934507.v1figshare

Additional file 3 of Monocyte-derived transcriptomes explain the ineffectiveness of abatacept in rheumatoid arthritis

10.6084/m9.figshare.26657821figshare

Additional file 5 of Monocyte-derived transcriptomes explain the ineffectiveness of abatacept in rheumatoid arthritis